The Wikipedia of Endodontics · by Dr. Ahmed Khaled
Create your free account
Dr.
👁
○ At least 8 characters
○ One capital letter
○ One number
Already have an account? Sign in
Welcome back
👁
Forgot password?
New here? Create a free account
Setting up your account...
EndoPedia
by Dr. Ahmed Khaled
Evidence-Based Learning
The Wikipedia of Endodontics
Cases · Anatomy · MCQs · Procedures · AI For students, GPs & specialists worldwide.
Dr. Ahmed Khaled
Endodontist · Cairo, Egypt
"Every answer in this app is sourced — not assumed."
Endo Fact of the Day
Loading...
⚡ Daily Challenge — 3 Questions
Free · Refreshes daily
📊 Your Challenge Stats
vs all users
0
Day Streak
—
Your Accuracy
—
Global Avg
All Modules
🗺️
Orifice Atlas
Accurate orifice maps per Vertucci
🔢
Nomenclature
FDI · Universal · Palmer
📝
MCQ Bank
Randomized · AAE-aligned bank
📋
Procedure Guides
Access · Irrigation · Obturation
🦷
Clinical Cases
Interactive · AAE-based diagnosis
🔍
Pulp Diagnosis
Symptoms → AAE diagnosis
🤖
Dr. Endo — AI Assistant
PROEvidence-graded answers
Trained on Cohen's Pathways 11th & 12th Ed., AAE guidelines, JOE, IEJ, EEJ, and landmark literature. Ask any clinical endo question.
Maxillary
Mandibular
Select tooth
Tooth Nomenclature
Three Numbering Systems
FDI — International
Two-digit. Quadrant (1=UR, 2=UL, 3=LL, 4=LR) + position 1–8. Used in all international journals.
16 = Upper right first molar
Universal — American (ADA)
Numbers 1–32 starting upper right 3rd molar, sweeping upper arch, then lower right to left.
#3 = Upper right first molar
Palmer — Clinical
Bracket symbol showing quadrant + number 1–8. Widely used in UK, Europe, and clinical notes.
⌐6 = Upper right first molar
Look Up Any Tooth
1. Quadrant
2. Tooth
FDI
—
International
UNIV.
—
American
PALMER
—
Clinical
Full Permanent Dentition
Tooth
FDI
Univ.
Palmer
MCQ Bank — Randomized
⚡
Session score
0/0
Q 1/5
📝
Unlimited Questions — Pro
Full randomized bank with topic filters, weak-area tracking, and timed exam simulation.
Procedure Guides
Protocols aligned with AAE Guidelines · ESE Position Statements · Cohen's Pathways 12th Ed.
Access Cavity Preparation
AAE Guidelines · 6 steps
▼
1
Rubber dam isolation
Mandatory before access. Reduces microbial contamination and prevents irrigant accidents — non-negotiable per AAE position statement. (Cohen's 12th Ed. Ch.8)
2
Initial penetration
High-speed round bur #4–6 through deepest fossa, directed toward the pulp chamber. Aim toward the pulp horn, not the root canals. (Cohen's 12th Ed. Ch.8)
DG-16 explorer probes all corners. Law of centrality: floor at CEJ level. Law of colour: floor always darker than walls. In maxillary first molars, MB2 lies mesial to the MB1–palatal line in 60–95% of cases. (Cohen's 12th Ed. Ch.8)
5
Straight-line access
Modify walls until files enter canals without contacting cavity walls. Reduces file fatigue, transportation, and ledging. (Cohen's 12th Ed. Ch.8)
6
Conservative / ninja access
Preserves pericervical dentin improving fracture resistance — but increases MB2 miss-rate. ESE 2022 position: use only with CBCT guidance and magnification. (EEJ 2022 · Chan et al.)
Irrigation Protocol
AAE Clinical Considerations · 5 steps
▼
1
Primary irrigant — NaOCl
2.5–5.25% NaOCl is the gold-standard primary irrigant. Dissolves organic tissue, broad antimicrobial spectrum. Minimum 20mL per canal per session. (Zehnder — JOE 2006)
2
EDTA — smear layer removal
17% EDTA final rinse for minimum 60 seconds. Chelates inorganic smear layer. Never mix directly with NaOCl — chlorate precipitate forms. (Cohen's 12th Ed. Ch.9)
3
Passive Ultrasonic Irrigation (PUI)
PUI significantly improves debridement — acoustic streaming disrupts biofilm in isthmi and lateral canals unreachable by needles. 30–60s per canal per cycle. (van der Sluis — IEJ 2007)
4
Final rinse sequence
NaOCl → EDTA 60s (activated) → final NaOCl flush → dry with paper points. CHX 2% only when specifically indicated — never mix directly with NaOCl (precipitate). (AAE Clinical Considerations 2023)
5
Safety — prevent extrusion
Side-vented needle placed 2mm short of WL minimum. Never bind the needle. NaOCl extrusion causes severe tissue necrosis — immediate saline flooding and urgent OMFS referral. (Cohen's 12th Ed. Ch.9)
Warm Vertical Compaction
Schilder Technique · 5 steps
▼
1
Master cone fit
GP cone matching master apical file size. Confirm tugback at exact working length. Verify with periapical radiograph — GP tip 0–0.5mm from radiographic apex. (Cohen's 12th Ed. Ch.10)
2
Sealer application
Thin coat on canal walls. AH Plus (gold standard for seal) or bioceramic sealers (BioRoot RCS — biocompatible, sets in moisture). Sealer is supplemental to GP, not a substitute. (Cohen's 12th Ed. Ch.10)
3
System B down-pack
Heat carrier at 200°C. Sear and compact to 5mm from WL in one firm stroke. Seals apical delta and lateral canals under heat and pressure. (Cohen's 12th Ed. Ch.10)
4
Thermoplastic backfill
Obtura III / Elements at 160°C. Inject in 3–4mm increments with vertical condensation between each. Continuous GP flow eliminates voids. (Cohen's 12th Ed. Ch.10)
5
Coronal seal — same visit
Place IRM or GIC base immediately after obturation. Bacteria penetrate unsealed canals within 30 days on average. Definitive restoration as soon as possible. (Torabinejad et al. — JOE 1990)
Working Length Determination
ESE Position Statement · 4 steps
▼
1
Radiographic estimate
Measure root length on paralleling-technique periapical radiograph. Estimated working length ± 2mm. (Cohen's 12th Ed. Ch.7)
2
Electronic apex locator
Multi-frequency EALs (Root ZX, Propex Pixi, Raypex 6) achieve 90–95% accuracy within ±0.5mm. Canal must be irrigated and free of blood for accurate readings. (Duran-Sindreu — IEJ 2012)
3
Radiographic verification
Confirm EAL reading radiographically. File tip should appear 0.5–1mm short of radiographic apex in fully developed teeth. (Kuttler 1955)
Retreatment · REP · Microsurgery · Trauma · Pharmacology Irrigation · NiTi Systems · Anatomy · Materials Unlimited access with Scholar or Elite
Obturation — Bioceramic Sealer + Single ConePRO
BioC Sealer · TotalFill · 6 steps
▼
1
Canal drying
Dry the canal with one paper point only — bioceramic sealers require residual moisture from dentin for hydraulic setting. Do NOT overdry.
2
Sealer application
Apply a thin coat of bioceramic sealer using a paper point or lentulo spiral to the apical third and canal walls.
3
Master cone selection
Select the matched master cone (same taper as the final rotary file used). Confirm tugback at WL before sealer application.
4
Cone insertion
Insert the sealer-coated master cone to WL in a single smooth motion. Avoid excessive apical pressure.
5
Sear and condense
Sear the cone at the orifice level with a heated plugger. Vertical condensation of the coronal GP — no spreader needed.
6
Coronal seal
Clean the access floor immediately. Place 2 mm GIC base + definitive composite restoration same day where possible. (Cohen's 12th Ed. Ch.9)
Non-Surgical Retreatment ProtocolPRO
Cohen's 12th Ed. Ch.8 · ESE Guidelines · 7 steps
▼
1
Assess cause of failure first
CBCT before retreatment: identify missed canals, short fills, VRF, or extraradicular pathology. Treating without identifying the cause leads to repeat failure.
2
Remove the coronal restoration
Remove crown/bridge/post to gain straight-line access. Ultrasonic vibration for post removal — do not use high-speed burs on posts.
3
Dissolve coronal GP
Apply solvent (chloroform or eucalyptol) on a paper point for 30 sec to soften the coronal GP before rotary retreatment files engage it.
4
Rotary retreatment files
ProTaper Retreatment D1 (coronal), D2 (middle), D3 (apical). Use continuous clockwise brushing motion — no apical force. Recapitulate with #10 K-file between each file.
5
Solvent + hand file apical segment
Apply solvent again. Use H-files to engage and pull apical GP remnants. Confirm patency with a #10 K-file to full WL.
6
Full irrigation protocol
Copious NaOCl + 17% EDTA x 1 min + final NaOCl. PUI activation. The canal must be cleaner than the original treatment.
7
Ca(OH)2 dressing for two-visit approach
For retreatment of infected cases: Ca(OH)2 dressing x 4 weeks before obturation. Obturate at visit 2. (Cohen's 12th Ed. Ch.8)
Regenerative Endodontic Procedure (REP)PRO
AAE REP Considerations 2021 · 8 steps
▼
1
Patient selection
Indication: necrotic immature permanent tooth with open apex. Confirm with CBCT (open apex, thin walls, periapical lesion).
2
Visit 1 — Access and irrigation
Under rubber dam: access, irrigate with ≤1.5% NaOCl only (higher concentrations kill SCAP). Final rinse: 17% EDTA x 1 min. Dry with paper points.
3
Antibiotic dressing
Anterior teeth: DAP (ciprofloxacin + metronidazole — no minocycline to avoid discolouration). Posterior: TAP acceptable. Place to 2 mm below CEJ. Seal with GIC.
4
Visit 2 — Re-irrigation
4 weeks later: remove dressing, irrigate with 17% EDTA only (no NaOCl at visit 2 — preserves SCAP). Dry with paper points.
5
Evoke bleeding
Pre-curve a #25 K-file, pass 3 mm beyond the WL to disrupt the apical papilla. Bleed to the CEJ level. Allow 15 min for blood clot formation.
6
Alternative scaffold if no bleeding
If bleeding cannot be evoked: use PRF membrane or collagen sponge as scaffold material introduced to the CEJ level.
7
MTA barrier
Place 3-4 mm of white MTA over the blood clot/scaffold using a MAP system. Moist cotton pellet over MTA. Seal access with GIC.
8
Final restoration
At the following visit: composite restoration. Monitor at 6, 12, 18, 24 months with PA. (AAE REP 2021)
Periapical Microsurgery ProtocolPRO
Cohen's 12th Ed. Ch.11 · Kim SG 2006 · 8 steps
▼
1
Pre-operative CBCT planning
Mandatory: assess lesion size, root anatomy, proximity to IAN/sinus/mental foramen. Plan osteotomy approach and flap design.
2
Flap design and reflection
Sulcular or Ochsenbein-Luebke flap. Full-thickness mucoperiosteal elevation. Identify and protect mental foramen.
3
Osteotomy
Round bur #6 or piezo tip under sterile saline irrigation. Create a bony window over the apex — minimum 4 mm diameter.
4
Curettage of lesion
Curette all granulation/cystic tissue. Submit for histopathology — mandatory. Achieve haemostasis with Racellet pellets + 1:50,000 epi.
5
Root-end resection
Resect 3 mm of the root apex perpendicular (0° bevel) to the long axis. Removes 93% of apical lateral canals and the entire apical delta.
6
Ultrasonic retropreparation
Under the surgical microscope: ultrasonic Class I cavity 3 mm deep, parallel to the long axis. Inspect for isthmus, cracks, and number of canals.
7
Retrograde fill — MTA or bioceramic
Dry the retroprep cavity. Deliver MTA or bioceramic putty in increments. Condense and verify complete 360° fill under the microscope before closing.
8
Flap closure
5-0 or 6-0 monofilament sutures. Interrupted pattern. Remove at 48-72 hours. Review at 6 months and 4 years. (Cohen's 12th Ed. Ch.11)
Refrigerant spray (Endo-Ice) on cotton pellet. Apply to middle third of facial surface. Normal: transient <5 sec. IRP: prolonged >30 sec. Necrotic: no response. Always compare to a control tooth.
2
Thermal test — heat
Heated GP stick or heated ball burnisher applied to the middle facial third (petroleum jelly barrier). Heat pain relieved by cold = late-stage IRP.
3
Electric pulp test (EPT)
Dry the tooth, toothpaste as conductor. Start at zero and increase slowly. Normal: mid-range. Early IRP: low threshold. Necrotic: no response at maximum setting.
4
Percussion and palpation
Percussion (mirror handle): tests PDL status, not pulp. Palpation (finger pad over apical mucosa): tests periapical bone. Always compare to contralateral.
5
Probing and selective anaesthesia
6-point probing for endo-perio lesions. Selective anaesthesia (IANB) when pain cannot be localised to an arch. Confirm all 4 tests before making pulp diagnosis. (AAE 2009)
Post-Endodontic Pain Management ProtocolPRO
AAE Pain Management 2022 · 5 steps
▼
1
First-line: NSAID + APAP combination
Ibuprofen 400 mg + paracetamol 500-1000 mg every 6 hours for 48 hours. Start BEFORE the local anaesthetic wears off (pre-emptive dosing). Outperforms opioids consistently in meta-analyses.
2
Pre-operative corticosteroid
For high flare-up risk (acute IRP): dexamethasone 4-8 mg orally 1 hour pre-operatively. Significantly reduces post-operative pain intensity.
3
Hot tooth — supplemental anaesthesia
IANB failure for IRP: PDL injection 0.2 mL lidocaine 2% + epi 1:100,000 at mesial and distal roots. Intraosseous as alternative. Intrapulpal as absolute last resort.
4
Antibiotic indication
Antibiotics are NOT analgesics. Prescribe only for systemic signs: fever >38°C, trismus, cellulitis, lymphadenopathy. First-line: amoxicillin 500 mg q8h x 5 days.
5
Metronidazole addition
Add metronidazole 400 mg q8h for spreading infections not responding to amoxicillin alone. Warn patient: no alcohol during course + 48h after. (AAE 2023)
Avulsion — Emergency Management ProtocolPRO
IADT Guidelines 2020 · 6 steps
▼
1
On-scene management
Do NOT scrub the root. Rinse gently with saline or milk. Ideal storage: HBSS. Next best: cold milk. Emergency: buccal vestibule. Time is critical — every minute counts.
Local anaesthesia. Saline socket irrigation. Gently replant with digital pressure. Flexible splint x 2 weeks. RCT within 7-10 days.
4
Open apex (<60 min)
Replant immediately. Revascularisation may occur — do NOT perform RCT unless infection signs develop. Monitor cold test and PA at 4 wk, 8 wk, 6 mo, 12 mo.
5
Non-viable PDL (>60 min dry, closed apex)
Soak in 2% sodium fluoride for 20 minutes. Ex-vivo RCT. Replant to maintain alveolar bone for future implant. Expect progressive replacement resorption.
6
Post-replantation monitoring
PA at 2 wk, 4 wk, 8 wk, 6 mo, 12 mo, annually x 5 years. Monitor for EIR, replacement resorption, and ankylosis. (IADT 2020)
CBCT — Indications and ProtocolPRO
AAE/AAOMR Position Statement 2015 · 5 steps
▼
1
Justification principle
CBCT is indicated ONLY when conventional radiography is insufficient AND the diagnostic information will change clinical management. Always document the clinical indication.
2
FOV selection
Always use the SMALLEST FOV that covers the area of interest (typically 4x4 cm or 5x5 cm for endodontic cases). Smaller FOV = higher resolution + lower dose.
Select a master cone that exactly matches the final rotary instrument taper. Confirm tugback at WL. No tugback = re-gauge and refit before proceeding.
2
Plugger pre-fitting
Pre-fit three pluggers at 3, 5, and 7 mm from WL. Working plugger should bind 3-5 mm short of WL. Never force to WL — risk of root fracture.
3
Sealer application
Coat the canal walls and master cone with a thin film of sealer using a paper point or lentulo. Insert the master cone immediately.
4
Down-pack (System B)
Apply heat burst at 200°C at the orifice level. Advance plugger in 1-2 mm increments with heat-cut-condense cycles. Final condensation 3-5 mm from WL.
5
Verify apical seal
Take a check radiograph after the down-pack. The apical fill should be homogeneous, void-free, and within 0.5-2 mm of the radiographic apex.
6
Backfill (thermoplastic injection)
Inject warm GP in 2-3 mm increments from the bottom of the down-pack space to the orifice. Condense each increment before the next injection.
7
Coronal seal
Clean the access floor of sealer and GP. GIC base 2 mm. Composite restoration. Document with PA. (Schilder 1967)
Vertical Root Fracture — Diagnosis ProtocolPRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Clinical triad
VRF triad: (1) isolated narrow deep probing pocket (buccal or lingual), (2) parulis/sinus tract adjacent to a treated tooth, (3) J-shaped or halo periradicular bone loss on imaging.
2
Percussion sound test
Tap with a mirror handle: metallic/high-pitched percussion note vs adjacent teeth = VRF or ankylosis sign. Compare bilaterally.
3
CBCT assessment
Halo sign: circumferential bone loss around the fractured root. CBCT is the most diagnostic non-invasive test — review axial and coronal slices for the fracture line.
4
Surgical exploration
When CBCT is inconclusive: reflect a full-thickness flap. The fracture line is often stained brown by endotoxins. Methylene blue staining may highlight the line.
5
Prognosis
Single-rooted tooth: hopeless — extraction. Multi-rooted molar: hemisection of the affected root may salvage the remaining sound root. (Cohen's 12th)
Internal: centred within the canal, enlarges canal outline, intact external surface. External: off-centre, originates from external root surface, does not respect the canal outline.
2
ECR — Heithersay classification
Class I: small, superficial. Class II: near the pulp, no canal involvement. Class III: extends into the coronal root canal. Class IV: involves the entire canal — usually non-restorable.
3
External inflammatory resorption (EIR)
EIR post-trauma: rapid ragged resorption from an infected necrotic pulp. Urgent RCT + Ca(OH)2 is the only way to stop progression. Every week of delay worsens the prognosis.
4
Replacement resorption (ankylosis)
Metallic percussion + loss of PDL space + bony fusion. Irreversible. In children: decoronation to preserve alveolar bone. In adults: extract when infraocclusion becomes significant.
5
Internal resorption — management
Non-perforating: RCT with warm obturation to fill the irregular cavity. Perforating: MTA orthograde or surgical repair. CBCT determines extent and restorability. (Cohen's 12th Ch.16)
Separated Instrument — Management ProtocolPRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Initial assessment
PA radiograph immediately. Note: position (coronal/middle/apical third), length, canal curvature, diameter, and whether the apical third can be instrumented around the fragment.
2
Non-surgical retrieval (coronal third, straight)
Ultrasonic trephination creates a trough around the coronal end. IRS kit or Masserann system grasps and removes. Best success in straight, coronal-third fragments.
3
Bypass (mid-root to apical third)
Pre-curve a #08 K-file to attempt bypass around the fragment. If bypass established: shape around it, irrigate, obturate beyond the fragment if possible.
4
Leave in situ
If retrieval and bypass both fail, fragment is in a well-instrumented canal with no periapical lesion: obturate to the fragment, monitor. Success comparable to bypass in low-risk cases.
5
Surgical removal
If all non-surgical options fail and fragment is associated with persistent periapical pathology: apical resection removes the fragment with the apex. Retrograde + MTA retrofill. (Cohen's 12th)
Canal Transportation — Prevention ProtocolPRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Understanding transportation
Files removing dentin from the outer wall of a curved canal straighten it artificially. Causes: ledges, zips, elbows, perforations. Prevention is far easier than management.
2
Glide path establishment
Always establish a patent, smooth glide path before rotary NiTi. Minimum: #10 K-file passively reaching WL with smooth in-out motion. Ideal: #15 K-file glide path confirmed.
3
Crown-down instrumentation
Work from coronal to apical third sequentially. Remove coronal interferences first. Reduces lateral pressure on files in the curved segment.
4
Anti-curvature filing
Hand files: direct pressure away from the outer curvature. In mesial roots of lower molars: file toward the buccal/lingual walls, away from the furcation danger zone.
5
Ledge bypass
Pre-curve a #08 K-file 30° at 1 mm from the tip. Watch-winding motion with EDTA lubricant. Patience — not force. Progressive enlargement past the ledge once bypassed. (Cohen's 12th)
Vital Pulp Therapy — Direct Pulp Cap ProtocolPRO
ESE VPT Position Statement 2019 · 5 steps
▼
1
Patient and tooth selection
Ideal DPC: carious or mechanical exposure <1 mm, bleeding controlled within 3-5 min with cotton pressure, no spontaneous pain, no periapical changes. Mature or immature vital tooth.
2
Haemostasis
Sterile cotton pellet moistened with saline applied for 3-5 minutes. Do NOT use electrosurgery or ferric sulfate on the pulp — causes tissue necrosis.
3
Material selection
MTA or Biodentine are first-line (75-95% success at 5 years). Ca(OH)2 (Dycal) acceptable but lower long-term success (50-70%). Never use ZOE directly on the pulp.
4
Placement technique
Apply 2-3 mm of MTA or Biodentine directly over the exposure under magnification. GIC base over the CSC. Restore same day with composite.
5
Follow-up monitoring
Cold test and PA at 6 weeks, 6 months, 1 year. Success: vital, asymptomatic, calcific bridge visible at 6-12 months. Failure: IRP or necrosis → proceed to RCT. (ESE 2019)
Endodontic Emergencies — Clinical ProtocolPRO
AAE Emergency Guidelines 2023 · 6 steps
▼
1
Triage and diagnosis
History, clinical tests (cold, EPT, percussion, palpation, probing), PA radiograph. Establish accurate AAE pulp and periapical diagnosis before any treatment.
2
Acute IRP — emergency pulpotomy
If single-visit RCT is not possible: emergency pulpotomy (remove coronal pulp + Ca(OH)2/cotton + IRM seal) provides excellent short-term pain relief. Schedule complete RCT within 2 weeks.
3
Hot tooth management
IANB first. If inadequate: PDL injection 0.2 mL x mesial and distal. Intraosseous injection as alternative. Intrapulpal injection as last resort once the chamber is accessed.
4
Acute apical abscess with swelling
Localised fluctuant: I&D + drain through the canal. Systemic signs (fever >38°C, trismus): amoxicillin 500 mg + metronidazole 400 mg q8h. Hospitalise if Ludwig's angina suspected.
5
Phoenix abscess (post-visit flare)
Re-enter the canal for drainage. Irrigate copiously. New Ca(OH)2 dressing. Antibiotics if systemic involvement. Reassure patient — predictable complication of necrotic tooth treatment.
6
Post-treatment instructions
Expected post-op soreness 24-72 hours is normal. Ibuprofen 400 mg + APAP 500 mg q6h. Soft diet. Contact details if swelling develops or pain worsens beyond 72 hours. (AAE 2023)
Endo-Perio Lesion — Diagnosis and TreatmentPRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Pulp test first — always
The single most important test: vital pulp = primary periodontal lesion. Non-vital pulp = primary endodontic lesion. Never treat without establishing pulp status first.
2
Primary endo lesion
Source: infected root canal. Signs: non-vital pulp, periapical lesion, possible lateral bone loss from lateral canals. Treatment: RCT first — perio component often resolves spontaneously.
3
Primary perio lesion
Source: periodontal disease. Signs: vital pulp, generalised pocketing consistent with periodontitis. Treatment: periodontal therapy only. No RCT.
4
Combined endo-perio lesion
Both pathologies coexist independently. Non-vital pulp + true periodontal pocket. RCT first, re-evaluate at 3 months. If perio component persists: periodontal surgery + GTR.
5
Palatogingival groove — special case
Upper lateral incisors: probe for a palatogingival groove. The groove creates a combined endo-perio lesion requiring RCT + surgical groove sealing (MTA/composite) + GTR. (Cohen's 12th Ch.7)
Smear Layer Removal — Irrigation ProtocolPRO
McComb & Smith 1975 · Pashley 1984 · 5 steps
▼
1
What is the smear layer?
A 1-5 µm layer of organic and inorganic debris created by instrumentation. Occludes dentinal tubule orifices, harbours bacteria, and prevents sealer penetration. Must be removed.
2
EDTA — inorganic component
17% EDTA chelates the inorganic calcium phosphate smear layer. Minimum contact time: 1 minute. Agitate with ultrasonic tip or EndoActivator during contact time.
3
Final NaOCl — organic component
After EDTA: final NaOCl flush removes the exposed organic matrix left after the inorganic component is dissolved by EDTA.
4
Sequence matters
Correct sequence: NaOCl throughout shaping → 17% EDTA x 1 min → final NaOCl flush → paper point dry → obturate. Never use EDTA and NaOCl simultaneously — they neutralise each other.
5
Ultrasonic activation enhances removal
PUI during the EDTA step significantly improves smear layer removal from lateral canals and isthmuses vs passive syringe irrigation alone. (Van der Sluis 2007)
NaOCl Accident — Emergency ProtocolPRO
Cohen's 12th Ed. Ch.9 · 5 steps
▼
1
Recognition
Sudden severe burning pain, rapid soft tissue swelling (sublingual, submandibular, infraorbital), patient distress, metallic taste, possible paraesthesia.
2
Immediate action
Stop irrigation immediately. Do NOT continue. Remove the rubber dam and assess the patient. Attempt gentle aspiration of the irrigant from the canal.
3
Pain and swelling management
Ice packs immediately (20 min on, 10 min off). Strong analgesics. Corticosteroids: dexamethasone 8 mg IM/IV to reduce swelling.
4
Antibiotic prophylaxis and monitoring
Amoxicillin 500 mg + metronidazole 400 mg q8h x 5 days. Monitor closely for airway compromise — hospitalise if swelling is progressive.
5
Documentation and prevention
Document fully. Prevention: side-vented needle, never binding, 2-3 mm short of WL, gentle syringe pressure. Confirm free needle movement before each irrigation. (Cohen's 12th Ch.9)
Antibiotic Stewardship in EndodonticsPRO
AAE Antibiotic Stewardship 2023 · 5 steps
▼
1
When NOT to prescribe
Localised abscess with drainage, symptomatic IRP, symptomatic SAP, reversible pulpitis, post-RCT soreness. Antibiotics cannot reach the avascular necrotic pulp.
A post retains a core, NOT the tooth. Needed only when <50% coronal structure remains. With ≥2 mm ferrule and ≥3 walls: post may not be needed.
2
Ferrule effect is critical
A 2 mm ferrule of sound coronal dentin at the crown margin is the single most important factor preventing root fracture. No ferrule = high fracture risk regardless of post type.
3
Fibre post vs metal post
Fibre posts: modulus of elasticity matches dentin (~18 GPa), distributes stress physiologically. Metal posts: 5-10x stiffer, concentrate stress → root fracture. Fibre posts are first choice.
4
Post length and diameter
Equal to crown length or 2/3 of root length. Leave 4-5 mm of apical GP seal. Diameter: as narrow as possible.
5
Timing of restoration
Permanent restoration within 1 month of obturation. Full crown for all RCT-treated posterior teeth. (Ray & Trope 1995)
Dental Trauma — Splinting GuidelinesPRO
IADT Guidelines 2020 · 5 steps
▼
1
When to splint
Subluxation (if mobile), extrusive luxation, lateral luxation, replanted avulsed teeth, alveolar fracture. NOT required for concussion or stable horizontal root fractures.
2
Flexible splinting
0.016 stainless steel wire + composite bonded to 2-3 teeth on either side of the injured tooth. Allows physiological movement to prevent ankylosis.
Cervical third root fractures and alveolar fractures: rigid immobilisation for 4 weeks (heavy stainless steel wire + acrylic or titanium trauma splint).
5
Monitoring during splinting
Cold test at splint removal. Non-vital at removal: initiate RCT immediately. PA at 4 wk, 8 wk, 6 mo, 12 mo, annually x 5 years. (IADT 2020)
Calcium Hydroxide Dressing ProtocolPRO
Cohen's 12th Ed. Ch.5 · 5 steps
▼
1
Indications
Necrotic teeth with periapical lesion (two-visit RCT), avulsion (arrests EIR), persistent exudate at visit 2.
2
Preparation
Ca(OH)2 powder + sterile water/saline to a creamy paste. Do NOT mix with eugenol — inactivates the hydroxyl ions.
3
Placement
Lentulo spiral at low RPM to within 1 mm of WL, or MAP system. Confirm with PA — must fill the full prepared canal length to be effective.
4
Duration and renewal
Most effective in first 1-4 weeks. Renew if delayed beyond 4 weeks. Long-term use >3 months weakens dentin (Doyon 2005). Maximum 3 months.
5
Removal
Copious NaOCl + size 35 K-file to mechanically disrupt Ca(OH)2. Final 17% EDTA x 1 min removes remnants. Confirm complete removal — Ca(OH)2 remnants impair sealer adhesion. (Cohen's 12th)
Electronic Apex Locator — Accuracy ProtocolPRO
4th Generation EAL · Cohen's 12th Ed. · 5 steps
▼
1
How 4th generation EALs work
Ratio method: two AC frequencies measure canal impedance. The ratio is constant at the minor foramen regardless of canal contents. Accuracy within 0.5 mm in >90% of cases.
2
Optimal canal conditions
Moist but not flooded: too dry = over-reading. Flooded with NaOCl = erratic OVER reading. One paper point to remove excess irrigant before measuring.
3
Clip position
Lip clip should contact moist lip mucosa, not dry lip or skin. Poor clip contact causes erratic readings.
4
Reading interpretation
EAL '0' = minor foramen (clinical WL). '0.5' = 0.5 mm short. 'OVER' = file is beyond the foramen. Confirm with a confirming PA film.
5
Special situations
Open apices: EAL unreliable — use paper point bleeding point or CBCT measurement. Perforations: EAL reads 'OVER' when file contacts moist tissue through a perforation. (Cohen's 12th)
Full Irrigation Protocol — Step by StepPRO
AAE · Boutsioukis 2010 · 6 steps
▼
1
NaOCl throughout shaping
Use 2.5-5.25% NaOCl throughout all shaping steps. Replenish after every 2-3 files — NaOCl is consumed by organic tissue and loses activity.
2
Side-vented needle technique
27-30 gauge side-vented needle, placed loose (non-binding) 2-3 mm short of WL. Never force the needle apically. Slow gentle plunger pressure.
3
Ultrasonic activation (PUI)
After shaping: flood canal with NaOCl, activate ultrasonic file (20-30 kHz) passively for 3 x 1-minute cycles. Most effective debridement method available.
4
EDTA — smear layer removal
17% EDTA x 1 minute minimum. Agitate with ultrasonic or sonic device. Do not use simultaneously with NaOCl — they neutralise each other.
5
Final NaOCl flush
After EDTA: final NaOCl flush to remove the exposed organic matrix.
6
Paper point drying
Dry with appropriate size paper points to WL. One paper point for bioceramic sealers (leave slightly moist). Two or more until fully dry for resin sealers. (Boutsioukis 2010)
Working Length — Combined EAL + Radiographic MethodPRO
Kuttler 1955 · AAE · 5 steps
▼
1
Anatomy reference
The apical constriction (CDJ/minor foramen) is the ideal terminus. It averages 0.5-1 mm short of the radiographic apex. The foramen exits at the radiographic apex in only ~17% of teeth.
2
Pre-flaring
Pre-flare the coronal and middle thirds BEFORE measuring WL. Removes the coronal dentinal triangle, improves EAL accuracy, and allows straighter file access.
3
EAL measurement
Flood canal with NaOCl. Advance #10 or #15 K-file until EAL reads '0' (minor foramen). Clinical WL = EAL '0' reading (retract to '0.5' = 0.5 mm short for the conservative approach).
4
Confirming radiograph
PA with file at the measured EAL WL. File should appear 0.5-2 mm short of the radiographic apex. Adjust only if significantly discrepant.
5
Reconfirm at obturation visit
After Ca(OH)2 treatment: always re-confirm WL with EAL — tissue changes may alter the measured length. (Kuttler 1955 · Cohen's 12th)
REP — Follow-up and Success CriteriaPRO
AAE REP 2021 · 5 steps
▼
1
Review schedule
PA at 6, 12, 18, and 24 months minimum. Annual PA thereafter for 5 years. Cold test at each visit. Primary success criterion: resolution of infection — not root development.
2
Radiographic success criteria
Complete: PAI 1-2, increased root length, increased wall thickness, apical closure. Partial: infection resolved, some root changes. Failure: persistent or new periapical lesion.
3
Positive cold test after REP
Return of cold sensitivity at 6-12 months = regeneration of pulp-like tissue. Rare but possible — confirms true pulp tissue regeneration.
4
If REP fails
At 12-18 months with no improvement: MTA apical plug (apexification) or apical surgery with retrofill. REP failure does not preclude subsequent apexification.
5
Root development expectations
Root lengthening and wall thickening occur in ~60-75% of successful REP cases. Apical closure in ~80% of successful cases at 24 months. (AAE REP 2021)
Time from perforation to repair is the strongest prognostic factor. Seal IMMEDIATELY with MTA.
3
MTA placement — orthograde
Identify the perforation with EAL (reads OVER). Dry with micro-suction or paper point. Deliver MTA with MAP system. Avoid MTA extrusion beyond the perforation.
4
Biodentine as alternative
Biodentine (12-min set vs 2.5h for MTA) is effective for furcal and cervical perforations where rapid setting is beneficial.
5
Surgical approach for cervical perforations
Cervical perforation communicating with the sulcus: MTA alone insufficient. Surgical flap + MTA + collagen membrane/GTR + primary closure. Prognosis is guarded. (Cohen's 12th Ch.9)
Endodontic Prognosis AssessmentPRO
Ørstavik 1996 · AAE · 5 steps
▼
1
Pre-operative factors
Periapical status is the strongest prognostic factor. No periapical lesion: ~90-95% success. With periapical lesion: ~75-85%.
2
Technical quality factors
Adequate preparation (length, shape, cleanliness), obturation quality, and coronal restoration quality. Ray & Trope (1995): coronal restoration quality > root filling quality for 4-year success.
RCT safe in all trimesters. Emergency treatment at any stage. Elective: second trimester preferred. LA: lidocaine + 1:100,000 epi safe at standard doses. Limit PA radiographs with shielding.
2
Diabetes mellitus
Well-controlled DM (HbA1c <8%): outcomes comparable to healthy patients. Poorly controlled: impaired healing, higher infection risk. More aggressive treatment + close follow-up.
3
Bisphosphonate patients
Prefer RCT over extraction. IV bisphosphonates (oncology): very high MRONJ risk — specialist referral for any surgical procedure.
4
Cardiac patients
Anticoagulants (warfarin INR <3.5): no modification for non-surgical RCT. IE prophylaxis: amoxicillin 2g pre-op for highest-risk patients (prosthetic valve, prior IE).
5
Immunocompromised patients
Neutropenia (WBC <1.5 x10^9/L): defer elective treatment. HIV (CD4 >200, HAART): standard RCT — outcomes comparable to immunocompetent. (Little JW)
Glide Path Establishment ProtocolPRO
Berutti 2009 · Cohen's 12th Ed. · 5 steps
▼
1
Definition and importance
A smooth, reproducible path from orifice to apical foramen. Without it, rotary NiTi files bind, deform, and separate. Most critical step before rotary instrumentation.
2
Hand file technique
#08 K-file: gentle watch-winding (90° CW, relax, advance 0.5 mm). Never force. Irrigate with NaOCl frequently. Once #08 reaches WL passively: advance to #10, then #15.
3
Patency filing
After each hand file: recapitulate with a #10 K-file to full WL to prevent apical debris compaction. This is the patency file — not a shaping file.
4
Mechanical glide path files
PathFile, ProGlider, or WaveOne Gold Glider after #10 hand file to WL. Expands the glide path to ISO 16-19 before the primary shaping file. Reduces rotary file stress significantly.
5
Confirmation
A glide path is confirmed when a #10 or #15 K-file slides passively to WL with minimal tactile resistance. Any binding = glide path NOT established — do not introduce rotary NiTi files. (Berutti 2009)
Reciprocating File Systems ProtocolPRO
WaveOne Gold · Reciproc · Cohen's 12th · 5 steps
▼
1
Principle of reciprocation
CCW cutting stroke (150°) engages and cuts dentin. CW release stroke (30°) disengages the file. Reduces angular fatigue per cycle — allowing safer use in curves.
2
WaveOne Gold system
Primary (25/.07): most canals. Small (20/.07): narrow/calcified. Medium (35/.06): larger canals. Large (45/.05): very large. One file per patient — single use only.
3
Motion technique
Slow, gentle in-out pecking motion (3-4 mm amplitude). Activate the motor BEFORE entering the canal. Withdraw and irrigate/recapitulate with #10 K-file every 3-4 cycles.
4
Checking for cyclic fatigue
Inspect the file after each use for unwinding, deformation, or bright spots. Single use only — do not re-sterilise and reuse reciprocating NiTi files.
5
When to use reciprocating vs rotary
Reciprocating: high curvature (>30°), narrow canals, high-risk cases. Continuous rotation: moderate curvature, established glide path, predictable anatomy. (Yared 2008)
RCT vs Implant — Decision FrameworkPRO
McGill 2008 · Cohen's 12th Ed. · 5 steps
▼
1
McGill Consensus
The natural dentition with successful endodontic treatment is the gold standard. An implant replaces a missing tooth — it is not a better alternative to a saveable tooth.
2
Evidence comparison
10-year survival: RCT + crown ~85-95%. Single implant ~90-95%. Comparable outcomes. RCT maintains proprioception, preserves alveolar bone, and is reversible.
3
When extraction is justified
Non-restorable crown-root ratio. Grade III furcation with >50% bone loss. Vertical root fracture. Persistent failure after adequate retreatment and surgery.
4
Alveolar bone preservation
If extraction is necessary: socket preservation graft immediately after extraction to maintain ridge dimensions for future implant placement.
5
Cost-effectiveness
RCT + crown is more cost-effective per tooth-year. Extraction + implant carries higher initial cost, maintenance costs, and surgical complication risks. (McGill 2008)
Mandibular First Molar — Anatomy GuidePRO
Cohen's 12th Ed. Ch.7 · De Pablo 2010 · 5 steps
▼
1
Root system
2 roots in >95%: mesial root (MB + ML canals, isthmus common) and distal root (1 or 2 canals). Radix entomolaris (3rd root, distolingual): 5% Europeans, up to 40% in some Asian groups.
2
Mesial root — key anatomy
MB and ML canals almost always present. Isthmus connects them — target for PUI. The furcal (inner) wall is dangerously thin (~0.2 mm). File away from the furcation (anti-curvature technique).
3
Distal root — large oval canal
Wide and oval in many cases. Two canals (DB + DL) in 30-40%. A large round access to the distal orifice alone may miss the distal-lingual canal. Probe with DG-16.
4
Access shape
Trapezoidal: wider mesially (MB + ML side by side), narrower distally. Extend the access to expose all orifices per Krasner & Rankow Laws of Symmetry.
5
Middle mesial canal
Present in ~15% of lower first molars. Located between MB and ML orifices. Negotiate with a pre-curved #08 K-file. Usually joins MB or ML apically. (De Pablo 2010)
Maxillary First Molar — Anatomy GuidePRO
Cohen's 12th Ed. Ch.7 · Krasner 2004 · 5 steps
▼
1
Root system
3 roots: mesiobuccal (MB), distobuccal (DB), palatal. The palatal root is the longest and largest. The MB root is the most curved and complex.
2
MB2 — the key canal
MB2 present in 60-95% of maxillary first molars. Located along the developmental groove from MB1 toward the palatal orifice. Technique: DG-16 explorer along the groove, then LN bur trough if not found.
3
Access shape
Rhomboidal: 4 corners for MB1, MB2, DB, and palatal. The access must be extended to expose MB2 — a small round access will miss it routinely.
4
Palatal root canal
Large, oval/round, often #40-60 at the apex. Easiest to negotiate but requires large shaping. Ultrasonic activation essential for the oval cross-section.
5
MB root danger zones
Curves sharply distolingually. Risk of transportation at the curvature. Use flexible NiTi, crown-down technique, and anti-curvature pressure. (Krasner 2004)
Crown-Down Instrumentation TechniquePRO
Cohen's 12th Ed. Ch.8 · Schilder 1974 · 5 steps
▼
1
Principle
Work from coronal to apical third in sequential steps. Remove coronal and middle third interferences first. Reduces force at the curve and prevents apical debris compaction.
2
Step 1 — Coronal pre-flaring
Gates Glidden #2 and #3 in the coronal third. Removes the dentinal triangle, provides straight-line access, and improves EAL accuracy. Never force GG burs — passive brushing outward.
3
Step 2 — Middle third
NiTi rotary files in the middle third until resistance is felt. Recapitulate with #10 K-file after each file. Irrigate copiously between files.
4
Step 3 — Measure WL
After coronal and middle third preparation, measure WL accurately with EAL + confirming PA. Coronal resistance is now gone, allowing a more accurate EAL reading.
5
Step 4 — Apical shaping
Advance through the apical third sequentially with copious irrigation and recapitulation between each file. Stop at the file that achieves the desired apical size. (Schilder 1974)
Managing Calcified CanalsPRO
Cohen's 12th Ed. Ch.4 · 5 steps
▼
1
Pre-operative assessment
CBCT mandatory for significantly calcified canals. Measure the distance from the occlusal/incisal reference to the calcified region.
2
Access modification
Conservative access. Use CBCT measurements as a guide — go to the calculated depth, then switch to ultrasonic. Never drill blindly beyond CBCT-guided depth.
3
Ultrasonic technique
Start-X #1 or #2 ultrasonic tip at LOW power. Work in a brushing motion following the dentinal colour change (yellow → grey → brown = canal).
4
Micro-tools for negotiation
#06 or #08 K-file pre-curved at 1 mm from the tip. Watch-winding motion. EDTA lubricant. Extreme patience — do not force any instrument.
5
Trephination as last resort
Only if all other methods fail and CBCT confirms the canal path. The risk of perforation is highest here — always confirm with CBCT-guided measurements. (Cohen's 12th)
Character (sharp/dull/throbbing), duration, triggers, timing (nocturnal/diurnal), previous treatment, relevant medical history.
2
Step 2 — Clinical examination
Extra-oral: asymmetry, swelling, lymphadenopathy. Intraoral: soft tissue, sinus tracts (trace with GP cone), visual inspection. 6-point probing.
3
Step 3 — Pulp tests
Cold → EPT → heat (if cold negative and spontaneous heat pain). Always test a control tooth first. Document: no response / normal / hyperresponsive / prolonged.
4
Step 4 — Periapical tests
Percussion (mirror handle). Palpation (finger pad over apical mucosa). Bite test (Tooth Slooth on each cusp for suspected CTS).
5
Step 5 — Radiographic assessment
PA radiograph. Angled PA for suspected extra canals. CBCT when indicated. Assign formal AAE pulp and periapical diagnosis before treatment. (AAE 2009)
Endodontic Flare-Up PreventionPRO
Cohen's 12th Ed. Ch.5 · 5 steps
▼
1
High-risk case identification
Pre-operative spontaneous pain + necrotic tooth with periapical lesion = highest flare-up risk. Two-visit protocol with Ca(OH)2 dressing recommended.
2
Pre-operative corticosteroid
Dexamethasone 4-8 mg orally 1 hour pre-operatively. Significantly reduces post-operative pain intensity in vital inflamed teeth (IRP). Single dose — no systemic adverse effect.
3
Meticulous debridement
Thorough mechanical and chemical debridement is the single most important prevention strategy. Copious NaOCl + EDTA + PUI activation.
4
Avoid over-instrumentation
Do not instrument beyond WL. Apical extrusion of debris is the most common cause of post-treatment flare-up.
5
Post-operative analgesia
Ibuprofen 400 mg + paracetamol 500-1000 mg BEFORE the LA wears off (pre-emptive). Continue q6h x 48h. Antibiotics only if systemic signs present. (Cohen's 12th)
Rubber Dam Isolation ProtocolPRO
AAE Position Statement 2016 · 5 steps
▼
1
Rubber dam is mandatory
Standard of care for all endodontic procedures per AAE, ESE, and ADA. Prevents NaOCl aspiration, maintains a sterile field, improves visibility, and protects the airway.
Use non-latex rubber dam (nitrile/vinyl) for documented latex allergy. Non-latex clamps also available. Document the allergy and dam material used.
4
Difficult isolation
Broken-down teeth: apply Oraseal/caulk around the dam margins. Build up the coronal structure with GIC before dam placement if needed.
5
Antimicrobial supplementation
After dam placement: scrub the access site with NaOCl or CHX before cutting through enamel. Decontaminates the surface microbiome before entering the pulp chamber. (AAE 2016)
Endodontic Outcome Measurement — PAI ScalePRO
Ørstavik PAI 1986 · ESE Quality Guidelines · 5 steps
▼
1
PAI scoring
PAI 1: normal. PAI 2: small bone changes. PAI 3: changes with mineral loss. PAI 4: well-defined radiolucency. PAI 5: severe periodontitis with exacerbating features.
2
Success criteria at 4 years
Complete healing: PAI 1-2 with no clinical symptoms = healed/success. Healing: progressive PAI reduction. Failure: PAI unchanged or worsening at 18-24 months.
3
Clinical success criteria
No pain, no swelling, no sinus tract, no loss of function, no probing defects, normal periapical structures on PA.
4
Success rates
Initial RCT without lesion: ~90-95%. With lesion: ~75-85%. Retreatment with lesion: ~65-75%. Periapical surgery (modern microsurgery): ~85-95%.
5
Documentation standard
Record pre-treatment and post-treatment PA with the same technique. Document PAI score at each review. Objective clinical records are essential for medicolegal protection. (Ørstavik 1986)
C-Shaped Canal System ProtocolPRO
Fan 2004 · Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Recognition and prevalence
Most common in mandibular second molars. Prevalence: ~8% in Caucasians, up to 44% in Chinese/Korean populations. Fan classification: Type I-III based on shape continuity.
2
CBCT pre-operatively
Always use CBCT for suspected C-shaped systems to map the ribbon extent and classify the type before treatment.
3
Access modification
The access must expose the full C-shaped orifice outline. An oval or C-shaped access is required — a round access will leave a portion of the ribbon unexposed.
4
Irrigation is the key treatment step
Round files cannot clean the connecting ribbon. PUI (3 x 1-min NaOCl cycles) + XP-endo Finisher are essential. The ribbon harbours biofilm unreachable by files.
5
Obturation
Cold lateral condensation is inadequate. Warm vertical condensation or carrier-based thermoplastic obturation can fill the irregular C-shaped cross-section. (Fan 2004)
Sinus Tract Tracing ProtocolPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Never assume the nearest tooth
The stoma may be far from the source tooth. A mandibular molar sinus tract can originate from a premolar. Always trace.
2
GP cone tracing technique
Insert a #25 or #30 GP cone gently into the stoma until resistance is felt. Take a PA with the cone in situ. The cone tip will be at the source tooth apex.
3
Multiple tracing
If the PA shows the cone tracks to an unexpected tooth: test the identified tooth with cold, EPT, and percussion. Confirm with CBCT if inconclusive.
4
What the sinus tract tells you
Confirms chronic apical abscess. The sinus tract is the drainage pathway. Closure after RCT = successful outcome. Persistent sinus tract at 3 months = failure.
5
Post-RCT sinus tract closure timeline
Most sinus tracts close within 2-6 weeks of adequate RCT. If still present at 3 months: suspect missed canal, microleakage, or extraradicular biofilm. (Cohen's 12th)
Obturation Quality AssessmentPRO
Cohen's 12th Ed. Ch.9 · 5 steps
▼
1
Radiographic assessment criteria
Homogeneous fill without voids, GP terminating 0.5-2 mm from the radiographic apex, appropriate taper, no sealer extrusion.
2
Void identification
Voids appear as radiolucencies within the GP fill. Apical third voids are most clinically significant. Warm techniques virtually eliminate voids.
3
Length assessment
Short fill (<2 mm from apex) in necrotic teeth: significant failure risk. Overfill (beyond apex): slight reduction in success; sealer extrusion less problematic than GP extrusion.
4
Immediate retreatment indications
GP clearly beyond apex by >2 mm, significant void in apical third, or underfill by >3 mm: consider immediate re-obturation before the patient leaves.
5
Post-obturation coronal seal
A well-sealed access is as important as the quality of the root filling. Recontamination occurs within 30 days without a proper coronal seal. (Torabinejad 1990)
Vital Pulp Therapy — Pulpotomy in Primary TeethPRO
AAPD Guidelines 2023 · 5 steps
▼
1
Indications
Deep carious exposure in a vital primary molar with controlled bleeding. No spontaneous pain, no furcal radiolucency, no internal resorption, at least 2/3 root length remaining.
2
Armamentarium
Rubber dam (mandatory). Round bur #6 high-speed for access. Sterile cotton pellets. MTA or Biodentine. GIC base. Stainless steel crown ideally placed at the same appointment.
3
Pulp removal technique
Remove the entire coronal pulp with a round bur or spoon excavator. Saline irrigation of the chamber. Cotton pellet pressure for 3-5 minutes until haemostasis.
4
Material placement
MTA or Biodentine directly over all radicular pulp stumps. 2-3 mm thickness. GIC base over the MTA same appointment. Crown preparation.
5
Crown placement
Stainless steel crown at the same appointment. Composite alone without SSC in primary molars has significantly higher failure rates. (AAPD 2023)
Mandibular Premolar Anatomy ChallengesPRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Canal configuration
Type I (single canal): ~65-70%. Type V (1-2, dividing apically): ~20%. Type II (2-1, merging): ~5%. Always take an angled PA to reveal potential bifurcation.
2
Identifying Type V
EAL: two different readings as the file advances. Two separate orifices visible on the floor. Two separate foramina on angled PA or CBCT. Both canals must be independently treated.
3
Three-rooted maxillary first premolar
Rare variant (~1%): three roots (MB, DB, palatal). CBCT confirms. All three roots must be located and treated.
4
Access shape for bifurcated lower premolar
An oval, buccal-lingual access extension reveals the lingual canal orifice. A small round access will miss the lingual orifice in Type V cases.
5
Obturation of Type V
Two separate working lengths, two matched master cones, or warm vertical condensation. Cold lateral condensation with a single cone will not seal both foramina. (Cohen's 12th)
Maxillary Anterior Teeth — Anatomy GuidePRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Central incisor
Single canal Type I (>95%). Triangular labiolingual access (MB, ML, incisal pulp horns). Canal is wide labiolingually — use oval master cone or warm technique.
2
Lateral incisor
Single canal Type I (~90%). Highest prevalence of dens invaginatus and palatal grooves. Always inspect the palatal surface carefully.
3
Canine
Single canal Type I (~97%). Longest root in the mouth (average 27 mm). Pre-curve files for the apical curvature. WL often longer than expected.
4
Access outline for all maxillary anteriors
Palatal surface, triangular shape. Remove all three pulp horns to prevent pink discolouration from retained pulp tissue remnants.
5
Discolouration prevention
After RCT: ensure all pulp horn tissue removed from crown. Avoid grey MTA or minocycline-containing TAP in aesthetic zone cases. (Cohen's 12th)
Mandibular Canine and Incisors — AnatomyPRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Key Clinical Point
Lower incisors: ~40% have two canals (Type III). The lingual canal is invisible on standard PA. Always take a mesially angled film. Lower canine: single canal in ~94%, one of the most predictable teeth.
2
Clinical Application
Lower anterior access: small, oval outline on the lingual surface. Negotiate with a #10-15 K-file. Confirm canal count with angled PA or CBCT before declaring single canal. PUI is essential for the narrow lingual canal.
Maxillary Second Molar — Key DifferencesPRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Key Clinical Point
MB2 less frequent (~60% vs 95% for first molar). Canal orifices are more compact and distal. The MB root is less curved. Access outline is smaller and more distal than for the first molar.
2
Clinical Application
Check for MB2 — still possible in ~60%. More compact access. Watch for fused roots (taurodontism). Confirm all orifices before shaping. (Cohen's 12th Ch.7)
Mandibular Second Molar — AnatomyPRO
Fan 2004 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
Highest prevalence of C-shaped canal systems among all teeth. In non-C-shaped cases: 2 roots (mesial + distal), similar to first molar. Radix entomolaris less common than in first molar.
2
Clinical Application
Always suspect C-shaped anatomy in Asian patients. Pre-op CBCT for second molars in Asian populations is strongly recommended. (Fan 2004)
Thermal Testing — Advanced InterpretationPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Key Clinical Point
Cold test: most clinically reliable pulp test. The 'prolonged' threshold varies: >10-15 seconds is a reasonable clinical cut-off for IRP. 'No response' = necrosis or calcific metamorphosis.
2
Clinical Application
Combine cold test with clinical history and percussion for accurate diagnosis. A single test in isolation is insufficient. Document the response objectively (seconds, not vague descriptors). (AAE 2009)
Electric Pulp Testing — Protocol and PitfallsPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Key Clinical Point
EPT stimulates A-delta sensory nerve fibres — it tests nerve function, not pulp health. A calcified canal may give false negatives. Recently traumatised teeth may give false negatives for weeks.
2
Clinical Application
Apply to a dry tooth surface. Use toothpaste as conductor. Test the control tooth first to calibrate the patient's response. Never diagnose necrosis based on EPT alone. (Cohen's 12th Ch.1)
Bite Test — Cracked Tooth ProtocolPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Key Clinical Point
Sharp pain ON biting that RELEASES immediately = cracked tooth syndrome (rebound pain). Pain that persists after biting = different origin. Tooth Slooth applied cusp-by-cusp identifies the offending cusp.
2
Clinical Application
Transillumination: apply fibre optic light to the buccal surface in a dark room. A crack appears as a dark line interrupting the light transmission. Wedging test also confirms cracks. (Cohen's 12th Ch.1)
Selective Anaesthesia — Diagnostic ProtocolPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Key Clinical Point
When pain cannot be localised to an arch: IANB of the lower arch. If pain stops immediately = lower arch source. If pain persists = upper arch source. Then narrow down by infiltrations.
2
Clinical Application
Start with the most likely arch based on patient history. An IANB that stops the pain definitively diagnoses a mandibular source. Then systematic infiltrations narrow it to a specific tooth. (Cohen's 12th Ch.1)
Periapical Radiograph — Technique and InterpretationPRO
Cohen's 12th Ed. Ch.3 · 5 steps
▼
1
Key Clinical Point
Paralleling technique (long-cone paralleling) is more accurate than bisecting angle for endodontic diagnosis. Use film holders. Standard endodontic series: PA + angled PA.
2
Clinical Application
Read systematically: crown → root canal system → root outline → PDL space (widening?) → lamina dura (continuous?) → periapical region (radiolucency? size?). Classify with PAI. (Cohen's 12th Ch.3)
Post-Treatment Monitoring ProtocolPRO
ESE Quality Guidelines 2006 · 5 steps
▼
1
Key Clinical Point
Minimum review schedule: 6 months, 12 months, 4 years. Standardised PA technique (same angulation) at each visit for accurate PAI comparison.
2
Clinical Application
At 4 years: PAI 1-2, asymptomatic = success. Document in the patient record. Discharge to routine dental care with the note to report symptoms. (ESE 2006)
Endodontic Treatment Planning AlgorithmPRO
Cohen's 12th Ed. · AAE · 5 steps
▼
1
Key Clinical Point
Step 1: establish the diagnosis. Step 2: assess restorability. Step 3: assess strategic value. Step 4: assess patient factors (medical, financial, motivation). Step 5: present all options with evidence-based prognosis.
2
Clinical Application
Never start RCT without confirming restorability. A non-restorable tooth with a perfect RCT has no long-term benefit — only delays the inevitable and wastes the patient's money. (Cohen's 12th)
Mandibular Anaesthesia — Advanced TechniquesPRO
Malamed SF · 5 steps
▼
1
Key Clinical Point
IANB is the primary technique but fails in ~15-20% of cases for routine procedures and up to 30% for IRP. Supplemental techniques: PDL, intraosseous (CCLAD/X-tip), intraseptal, intrapulpal.
2
Clinical Application
PDL injection: 0.2 mL at 30° angle at mesial and distal. Slow pressure (30 seconds per injection). Effective within 30 seconds. Intraosseous (CCLAD): deposits LA directly in cancellous bone — profound anaesthesia. (Malamed)
Coronal Microleakage — Prevention ProtocolPRO
Torabinejad 1990 · Ray & Trope 1995 · 5 steps
▼
1
Key Clinical Point
Recontamination of the root canal system after obturation occurs within 30 days without an adequate coronal seal. The quality of the coronal restoration is a stronger predictor of 4-year success than root filling quality.
2
Clinical Application
Place a minimum 3-4 mm GIC or composite access fill immediately after obturation. Schedule permanent restoration (crown for posterior teeth) within 1 month. Inform the patient of this critical timeline. (Ray & Trope 1995)
Apical Gauging TechniquePRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Key Clinical Point
Apical gauging determines the true apical diameter — the size of the first file that binds snugly at WL. The master apical file (MAF) should be 2-3 sizes larger than the first binding file for adequate apical preparation.
2
Clinical Application
After WL measurement: sequentially insert K-files until one binds at WL. The size of the first binding file = the natural apical diameter. Select a MAF that is at least 2 sizes larger than the binding file. (Cohen's 12th)
1% NaOCl: lowest effective antimicrobial concentration. 2.5-3%: clinical standard. 5.25%: maximum concentration. Higher concentrations = greater tissue dissolution but more toxic. Volume is more important than concentration.
2
Clinical Application
Warm NaOCl (37-45°C) at 1% is as effective as room-temperature 5.25% for tissue dissolution (Sirtes 2005). Pre-warming is a simple upgrade. Never warm beyond 45°C. (Boutsioukis 2010)
Sonic Irrigation — EndoActivator ProtocolPRO
EndoActivator · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
EndoActivator operates at 1-6 kHz (sonic) — less powerful than ultrasonic (25-30 kHz) but safer (less risk of file separation or perforation). Most effective for biofilm disruption in the coronal and middle thirds.
2
Clinical Application
Use a medium or large tip for canals prepared to F2 or larger. Activate with 1 cm up-down motion for 3 x 30-second cycles per irrigant. Do not touch the canal walls. Final rinse with NaOCl after activation. (EndoActivator IFU)
XP-endo Finisher — ProtocolPRO
FKG Dentaire · 5 steps
▼
1
Key Clinical Point
MaxWire alloy: martensitic at room temperature (flexible), austenitic at body temperature (expands to 1 mm diameter). Contacts the untouched buccal and lingual walls of oval canals missed by round rotary files.
2
Clinical Application
After final shaping: activate XP-endo Finisher at 800-1000 RPM, torque 1 Ncm, in NaOCl for 60 seconds. Use a 1-2 cm in-out motion. Final irrigation sequence follows. Significant improvement in oval canal debridement vs rotary alone.
Crown-down: SX → S1 → S2 → measure WL → F1 → F2 → F3 (if needed). Never use PTG files without a confirmed glide path. Continuous clockwise rotation, 300 RPM, 2-3 Ncm torque. One file per patient for F1-F3 (Dentsply Sirona recommendation).
WaveOne Gold — Clinical ProtocolPRO
Dentsply Sirona · 5 steps
▼
1
Key Clinical Point
WaveOne Gold Small (20/.07), Primary (25/.07), Medium (35/.06), Large (45/.05). Reciprocating motion — one file per patient.
2
Clinical Application
Establish glide path (#10-15 K-file to WL). Activate WaveOne Gold motor program (400 RPM equivalent reciprocation). Gentle pecking motion 3-4 mm amplitude. Withdraw every 3-4 pecks, wipe the file, recapitulate with #10 K-file, irrigate. (WaveOne Gold IFU)
Reciproc Blue — Clinical ProtocolPRO
VDW · 5 steps
▼
1
Key Clinical Point
Reciproc Blue R25 (25/.08): most cases. R40 (40/.06): larger canals. R50 (50/.05): very large canals. Blue heat treatment improves flexibility compared to original Reciproc.
2
Clinical Application
Glide path mandatory (#10 K-file to WL minimum). VDW.SILVER reciproc motor programme. 3-4 mm gentle pecking amplitude. Never force apically. Withdraw and recapitulate with #10 K-file every 3-4 pecking cycles. One file per patient. (VDW IFU)
Bioceramics in Endodontics — OverviewPRO
Cohen's 12th Ed. · 5 steps
▼
1
Key Clinical Point
Calcium silicate cements (CSCs): MTA, Biodentine, BioAggregate, BioC Sealer, TotalFill. All share: high pH on setting (~12-13), calcium ion release, and cementum/bone regeneration stimulation.
2
Clinical Application
Applications: pulp caps, pulpotomies, apexification (MTA plug), REP (MTA barrier), perforation repair, retrograde fill, sealer (BioC Sealer). The most biologically active class of endodontic materials. (Cohen's 12th)
MTA — Clinical Handling ProtocolPRO
ProRoot MTA · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
Mix ratio: 3:1 powder to liquid (distilled water). Consistency: sand-castle texture (not too wet or too dry). Setting time: 2.5-4 hours in the presence of moisture.
2
Clinical Application
Delivery: MAP system in 2 mm increments. Condense with a moistened micro-plugger. Place a moist cotton pellet over the MTA and temporise. Review at next visit to confirm setting. Grey MTA: risk of discolouration — use white MTA or Biodentine in aesthetic zones. (Cohen's 12th)
Biodentine — Clinical Handling ProtocolPRO
Septodont · 5 steps
▼
1
Key Clinical Point
Capsule system: mix for 30 seconds in a triturator. Setting time: 12 minutes (initial) — significantly faster than MTA. Working time: 6 minutes.
2
Clinical Application
Deliver with a MAP system or a plastic instrument. Condense in increments. Place a moist cotton pellet over Biodentine. Can restore directly over set Biodentine with composite (no GIC intermediate needed). Less discolouration risk than grey MTA — use in aesthetic zones. (Septodont IFU)
AH Plus bonds to dentin amino groups (covalent bond) — the best adhesive of all sealers. Does NOT contain eugenol — composite restoration is safe. Most extensively studied sealer — positive control in most comparative research. (Cohen's 12th)
Gutta-Percha — Properties and HandlingPRO
Cohen's 12th Ed. Ch.9 · 5 steps
▼
1
Key Clinical Point
Composition: ~20% GP polymer, ~65-70% zinc oxide, ~10% barium sulfate (radiopaque), ~1-2% wax/antioxidants. Two crystalline phases: alpha (thermoplastic) and beta (room temperature, used in cones).
2
Clinical Application
GP cannot be autoclaved (deforms). Disinfect with 5.25% NaOCl x 1 min or 70% IPA before use. Store in cool, dark conditions. Check cones for brittleness before use — brittleness indicates degradation. (Cohen's 12th)
Silver Points — Historical Context and RetreatmentPRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Key Clinical Point
Silver points (introduced 1930s-1970s): unable to adapt to the oval canal cross-section and corrode over time (silver sulfide formation causes microleakage). No longer indicated for obturation.
2
Clinical Application
Silver point retreatment: ultrasonic vibration to break the coronal seal. Stieglitz or Masserann forceps to grasp and withdraw. The apical portion may have fused to dentin — bypass is the alternative if retrieval fails. (Cohen's 12th Ch.8)
Carrier-Based Obturation — Thermafil ProtocolPRO
Dentsply Sirona · 5 steps
▼
1
Key Clinical Point
Thermafil: plastic carrier coated with alpha-phase GP. The Thermafil Verifier confirms the apical preparation size before the heated obturator is introduced.
2
Clinical Application
Oven: 15 seconds for #25 carrier. Insert to WL in a single motion — do not hesitate once inserted. Sear the carrier at the orifice with a heated plugger. Cut the plastic shank. Backfill with additional warm GP or composite. (Dentsply Sirona IFU)
Endodontic Sealer Types — ComparisonPRO
Cohen's 12th Ed. Ch.9 · 5 steps
▼
1
Key Clinical Point
ZOE sealers (Pulp Canal Sealer): most soluble, most shrinkage, easiest to remove in retreatment. Epoxy resin (AH Plus): best adhesion, minimal shrinkage, moderate retreatability. Bioceramic (BioC): best biocompatibility, hardest to remove. GIC-based: hydrophilic, poor adhesion to GP.
2
Clinical Application
Select the sealer based on the clinical situation. AH Plus: best evidence, easiest retreatment balance. Bioceramic: primary obturation in ideal anatomy (single cone). ZOE: least preferred — high shrinkage. (Cohen's 12th Ch.9)
Understanding the historical context helps evaluate current evidence. Schilder's principles — continuously tapering funnel, smallest cross-section apically, original shape maintained — remain the gold standard regardless of technique. (Cohen's 12th)
Endodontic Instruments — Metallurgy OverviewPRO
Cohen's 12th Ed. Ch.8 · 5 steps
▼
1
Key Clinical Point
Standard NiTi (nitinol): austenitic at body temperature. M-Wire: austenitic + martensitic mixture — more flexible. CM Wire (controlled memory): fully martensitic — very flexible, shape memory. Gold/Blue heat treatment: austenitic but phase-shifted — increased flexibility, lower cyclic fatigue.
2
Clinical Application
Gold/Blue/R-Phase alloys have significantly lower cyclic fatigue than standard NiTi in severe curvatures. Choose heat-treated alloys for canals with curvature >20°. (Cohen's 12th)
Cyclic Fatigue — Prevention and ManagementPRO
Cohen's 12th Ed. · 5 steps
▼
1
Key Clinical Point
Cyclic fatigue: repeated tension-compression cycles at the point of maximum curvature accumulate metal fatigue until fracture. No torsional engagement is needed — the file fractures even when not binding.
2
Clinical Application
Prevention: heat-treated alloys (Gold, Blue, CM), reduce canal curvature with crown-down, limit number of uses, inspect files after each use, use reciprocating motion to reduce angular accumulation. (Cohen's 12th)
Torsional Fatigue and File Fracture PreventionPRO
Cohen's 12th Ed. · 5 steps
▼
1
Key Clinical Point
Torsional fracture: the file tip is locked in the canal while the shank continues rotating. Risk: narrow canals, files used without a glide path, aggressive apical pressure.
2
Clinical Application
Prevention: always establish a glide path, never force rotary NiTi files apically, respect the manufacturer's recommended torque setting, withdraw and recapitulate frequently, inspect files after each use. (Cohen's 12th)
Sodium Hypochlorite — Properties and OptimisationPRO
Zehnder 2006 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
NaOCl is the only irrigant that dissolves both vital and necrotic organic tissue. Active component: hypochlorous acid (HOCl). Consumed by organic matter — must be replenished. Volume > concentration.
2
Clinical Application
Warm NaOCl (37-45°C) significantly improves tissue dissolution. Side-vented needle 2-3 mm short of WL. Maximum safe concentration: 5.25%. For hypersensitive patients: 1-2% still effective when combined with PUI. (Zehnder 2006)
Chlorhexidine — Role in EndodonticsPRO
Cohen's 12th Ed. Ch.5 · 5 steps
▼
1
Key Clinical Point
CHX: broad-spectrum antimicrobial, substantivity (binds to hydroxyapatite and releases slowly for up to 12 weeks). Cannot dissolve organic tissue — NOT a replacement for NaOCl.
2
Clinical Application
Use as a final rinse in retreatment cases (for E. faecalis coverage). IMPORTANT: CHX + NaOCl = orange precipitate (para-chloroaniline) — a cytotoxic reaction. Separate with a saline flush between NaOCl and CHX. (Cohen's 12th)
EDTA — Properties and Correct UsePRO
Pashley 1984 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
17% EDTA: chelates calcium ions from the inorganic smear layer component. Contact time: 1 minute minimum. Does NOT have antimicrobial properties — purely a chelating agent.
2
Clinical Application
Use AFTER final NaOCl shaping, not throughout. Agitate with ultrasonic. Follow immediately with a final NaOCl rinse to remove the exposed organic matrix. Do not use EDTA and NaOCl simultaneously — the chlorine is neutralised. (Pashley 1984)
PUI: ultrasonic file oscillates freely (passively — not engaging the walls) in a canal flooded with irrigant. Creates acoustic streaming (fluid movement) and cavitation (microbubble formation).
2
Clinical Application
Technique: flood the canal with NaOCl. Insert the ultrasonic file (D1 or endo tip) without engaging the canal walls. Activate at 20-30 kHz for 3 x 1-minute cycles. Agitation drives irrigant into fins, isthmuses, and lateral canals. (Van der Sluis 2007)
Apical Negative Pressure Irrigation — EndoVacPRO
Boutsioukis 2010 · 5 steps
▼
1
Key Clinical Point
EndoVac: negative pressure draws irrigant apically via a micro-cannula suction tip rather than positive pressure delivery. Virtually eliminates the risk of NaOCl extrusion beyond the apex.
2
Clinical Application
Technique: macro-cannula for coronal third, micro-cannula (0.32 mm) placed at WL for apical third. Simultaneously dispense NaOCl from a needle in the coronal chamber while the suction draws it apically. Follow with PUI. (Boutsioukis 2010)
Smear Layer — Is Removal Beneficial?PRO
McComb 1975 · Pashley 1984 · 5 steps
▼
1
Key Clinical Point
Removal: allows better sealer penetration into dentinal tubules, eliminates bacteria harboured in the smear layer, and improves the sealer-dentin bond. The majority of evidence supports removal.
2
Clinical Application
Counterargument: some argue the smear layer acts as a physical barrier preventing bacteria from re-entering tubules. However, the consensus based on systematic reviews supports smear layer removal for optimal obturation quality. (Cohen's 12th)
Persistent infection (post-treatment disease): Enterococcus faecalis is the most commonly recovered species — resistant to Ca(OH)2 due to its proton pump. Also: Candida albicans (fungal). These require CHX as a final rinse in retreatment. (Nair 2006)
Biofilm in Root Canals — Clinical ImplicationsPRO
Ricucci & Siqueira 2010 · 5 steps
▼
1
Key Clinical Point
Biofilm bacteria are 100-1000x more resistant to antimicrobials than planktonic bacteria. The extracellular polymeric matrix physically protects embedded cells. NaOCl disrupts the matrix.
2
Clinical Application
Clinical implication: passive irrigation with NaOCl is insufficient to disrupt biofilm. Active agitation (PUI, EndoActivator, XP-endo Finisher) is essential. This is why multi-visit treatment with Ca(OH)2 further reduces biofilm load. (Ricucci 2010)
Enterococcus faecalis — Why It PersistsPRO
Sundqvist 1998 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
E. faecalis resists Ca(OH)2 via a proton pump that maintains intracellular pH against external alkalinity. It can survive as a monoinfection in treated root canals and cause post-treatment disease.
2
Clinical Application
Management: 2% CHX final rinse in retreatment cases (for E. faecalis substantive coverage). Ca(OH)2 alone is insufficient. CHX + NaOCl interaction: precipitate formation — always separate with a saline flush. (Sundqvist 1998)
Periapical Lesion — PathogenesisPRO
Nair 2006 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
The periapical lesion is the host immune response to bacterial toxins (LPS, lipoteichoic acid) leaking from the infected root canal. It is NOT the bacteria themselves — it is the immune reaction.
2
Clinical Application
Implication: eliminate the intracanal infection and the periapical immune response resolves on its own. This is why RCT heals periapical lesions. The bone regeneration that follows represents resolution of the immune lesion. (Nair 2006)
Apical Delta Anatomy — Clinical SignificancePRO
Kuttler 1955 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
The apical delta: multiple small accessory canals branching from the main canal in the apical 3 mm. Present in ~75% of teeth. These cannot be mechanically instrumented.
2
Clinical Application
Clinical significance: the apical delta is the primary reason for using chemical disinfection (NaOCl + PUI) rather than relying on mechanical preparation alone. Periapical surgery resects this complex (3 mm resection removes ~93% of the delta). (Kuttler 1955)
Lateral and Accessory Canals — SignificancePRO
Cohen's 12th Ed. · 5 steps
▼
1
Key Clinical Point
Lateral canals: present throughout the root at the mid-root level. Furcal canals: open into the furcation floor (up to 46% of multi-rooted teeth). All are potential portals of exit for pulpal disease.
2
Clinical Application
Lateral canals cannot be mechanically cleaned — chemical disinfection (NaOCl) and obturation with warm techniques that drive sealer and GP into them are essential. A lateral canal visible as sealer-filled on the PA is a positive finding. (Cohen's 12th)
Cementodentinal Junction — Why It MattersPRO
Kuttler 1955 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
The CDJ (cementodentinal junction) is the internal anatomical endpoint of the root canal at the junction of cementum and dentin. It corresponds to the apical constriction and averages 0.5-1 mm from the radiographic apex.
2
Clinical Application
Prepare and obturate to the CDJ for the best tissue response and seal. Working short of the CDJ leaves untreated canal space. Working beyond it creates a periapical irritant. The EAL '0' reading identifies the minor foramen at the CDJ. (Kuttler 1955)
Pulp Anatomy — Development and Clinical ApplicationPRO
Cohen's 12th Ed. Ch.7 · 5 steps
▼
1
Key Clinical Point
The pulp chamber reflects the external crown form — where the enamel rises, the pulp horn follows. Knowledge of the pulp horn positions prevents perforation during access preparation.
2
Clinical Application
The pulp recedes throughout life with secondary dentin deposition: the older the patient, the narrower the chamber. Elderly patients and heavily restored teeth need CBCT before difficult access preparation. (Cohen's 12th)
Dental Pulp Stem Cells — Clinical RelevancePRO
Gronthos 2000 · Cohen's 12th · 5 steps
▼
1
Key Clinical Point
Dental pulp stem cells (DPSCs) and stem cells of the apical papilla (SCAP) are the regenerative cell populations in vital pulp therapy and REP respectively.
2
Clinical Application
SCAP survive pulp necrosis because they have their own blood supply from the periapical tissues — the reason REP is feasible. Protecting SCAP during REP (low-concentration NaOCl, 17% EDTA final rinse) is the biological rationale for the gentle irrigation protocol. (Gronthos 2000)
Pulp Inflammation — HistopathologyPRO
Cohen's 12th Ed. Ch.1 · 5 steps
▼
1
Key Clinical Point
Reversible pulpitis: hyperaemia, initial inflammation confined to near the stimulus — no necrosis. Irreversible pulpitis: irreversible cell damage, inflammation extends deeper, internal pressure rises. Necrosis: liquefactive necrosis, no vital cells remaining.
2
Clinical Application
The histological state cannot be reliably determined from clinical tests alone. 'Irreversible pulpitis' as a clinical diagnosis means the pulp cannot be maintained as a healthy state — not necessarily confirmed histologically. (Cohen's 12th Ch.1)
Periapical diagnoses: Normal apical tissues / Symptomatic apical periodontitis / Asymptomatic apical periodontitis / Chronic apical abscess / Acute apical abscess. Both pulp AND periapical diagnoses must be recorded for every tooth. (AAE 2009)
AAE Classification 2009 — Periapical DiagnosesPRO
AAE Consensus Conference 2009 · 5 steps
▼
1
Key Clinical Point
Normal apical tissues: no symptoms, no radiographic changes. SAP: pain to percussion/palpation with or without lesion. AAP: radiolucency, no symptoms. Chronic apical abscess: sinus tract + non-vital tooth, minimal symptoms. Acute apical abscess: rapid onset pain, swelling, systemic signs.
2
Clinical Application
Assign both a pulp and periapical diagnosis for every tooth requiring endodontic evaluation. This is the foundation of the treatment decision. (AAE 2009)
Review your own cases at 6 months and 4 years. Compare the pre-treatment and post-treatment PA using the same angulation. Calculate your personal PAI improvement rate.
2
Clinical Application
A personal audit rate of PAI 1-2 at 4 years >85% is the target for cases without pre-operative periapical lesion. With lesion: >75%. Regular self-audit is the most powerful driver of clinical improvement and medicolegal protection. (ESE 2006)
Clinical Cases
Diagnoses aligned with the AAE/ESE 2025 Pulpal & Periapical Classification.
Swelling related to mandibular first molar
Easy
👤 35M🦷 FDI 46
35-year-old male presents with severe spontaneous pain for 3 days, with facial swelling and fever. Tooth 46. No response to cold or electric pulp testing. Diffuse facial swelling, tender submandibular lymph nodes, pain to percussion and palpation. Periapical radiolucency present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. No response to pulp testing = Pulp necrosis. Systemic signs (fever, diffuse facial swelling, lymphadenopathy) with a periapical lesion of pulpal origin = Apical periodontitis with systemic involvement (2025 term, replacing "acute apical abscess" with systemic spread).
Management: Immediate endodontic intervention to drain the infection (through the canal ± incision and drainage). Antibiotics indicated. Analgesics as needed. Urgent referral/hospital if airway is threatened.
Cold-sensitive maxillary first molar
Easy
👤 27F🦷 FDI 16
27-year-old female reports sharp pain to cold drinks that disappears within a second or two. Tooth 16. Heightened but non-lingering response to cold; normal to EPT. No tenderness to percussion or palpation. Moderate occlusal caries with sound dentine still visible between the caries and pulp on the radiograph. No apical changes.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A heightened but non-lingering cold response with caries approaching (not reaching) the pulp and no percussion tenderness fits Mild pulpitis. Apical tissues are clinically normal.
Management: Caries removal and restoration; vital pulp treatment only if exposure occurs. Root canal treatment is unlikely to be required.
Gum boil near maxillary lateral incisor
Medium
👤 41M🦷 FDI 12
41-year-old male notices a small recurring "pimple" on the gum above tooth 12, with little or no pain. No response to pulp testing. No or mild percussion tenderness. A gutta-percha cone traced from the sinus tract points to the apex of tooth 12 on the radiograph, where a periapical radiolucency is present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. A necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the AAE/ESE 2025 system (this replaces the older "chronic apical abscess").
Management: Root canal treatment. The sinus tract usually resolves once the intracanal infection is eliminated; review radiographically for healing.
🔒
Unlock Unlimited Cases
Diagnosis · Treatment planning · Complex anatomy Trauma · Retreatment · Surgery decisions Unlimited access with Scholar or Elite
Cold-sensitive maxillary central incisor
Easy
👤 18M🦷 FDI 11
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 11 (maxillary central incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary lateral incisor
Easy
👤 23F🦷 FDI 12
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 12 (maxillary lateral incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary canine
Easy
👤 28M🦷 FDI 13
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 13 (maxillary canine). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first premolar
Medium
👤 33F🦷 FDI 14
33-year-old female presents with a draining gum boil but no pain. Tooth 14 (maxillary first premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second premolar
Medium
👤 38M🦷 FDI 15
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 15 (maxillary second premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first molar
Medium
👤 43F🦷 FDI 16
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 16 (maxillary first molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second molar
Hard
👤 48M🦷 FDI 17
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 17 (maxillary second molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 41 (mandibular central incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular lateral incisor
Easy
👤 58M🦷 FDI 42
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 42 (mandibular lateral incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular canine
Easy
👤 63F🦷 FDI 43
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 43 (mandibular canine). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first premolar
Medium
👤 68M🦷 FDI 44
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 44 (mandibular first premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second premolar
Medium
👤 73F🦷 FDI 45
73-year-old female presents with a draining gum boil but no pain. Tooth 45 (mandibular second premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first molar
Medium
👤 18M🦷 FDI 46
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 46 (mandibular first molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second molar
Hard
👤 23F🦷 FDI 47
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 47 (mandibular second molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary central incisor
Easy
👤 28M🦷 FDI 11
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 11 (maxillary central incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 12 (maxillary lateral incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary canine
Easy
👤 38M🦷 FDI 13
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 13 (maxillary canine). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first premolar
Medium
👤 43F🦷 FDI 14
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 14 (maxillary first premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second premolar
Medium
👤 48M🦷 FDI 15
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 15 (maxillary second premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first molar
Medium
👤 53F🦷 FDI 16
53-year-old female presents with a draining gum boil but no pain. Tooth 16 (maxillary first molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second molar
Hard
👤 58M🦷 FDI 17
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 17 (maxillary second molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular central incisor
Easy
👤 63F🦷 FDI 41
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 41 (mandibular central incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular lateral incisor
Easy
👤 68M🦷 FDI 42
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 42 (mandibular lateral incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 43 (mandibular canine). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first premolar
Medium
👤 18M🦷 FDI 44
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 44 (mandibular first premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second premolar
Medium
👤 23F🦷 FDI 45
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 45 (mandibular second premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first molar
Medium
👤 28M🦷 FDI 46
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 46 (mandibular first molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second molar
Hard
👤 33F🦷 FDI 47
33-year-old female presents with a draining gum boil but no pain. Tooth 47 (mandibular second molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary central incisor
Easy
👤 38M🦷 FDI 11
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 11 (maxillary central incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary lateral incisor
Easy
👤 43F🦷 FDI 12
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary canine
Easy
👤 48M🦷 FDI 13
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 13 (maxillary canine). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 14 (maxillary first premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second premolar
Medium
👤 58M🦷 FDI 15
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 15 (maxillary second premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first molar
Medium
👤 63F🦷 FDI 16
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 16 (maxillary first molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second molar
Hard
👤 68M🦷 FDI 17
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 17 (maxillary second molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular central incisor
Easy
👤 73F🦷 FDI 41
73-year-old female presents with a draining gum boil but no pain. Tooth 41 (mandibular central incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular lateral incisor
Easy
👤 18M🦷 FDI 42
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 42 (mandibular lateral incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular canine
Easy
👤 23F🦷 FDI 43
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 43 (mandibular canine). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first premolar
Medium
👤 28M🦷 FDI 44
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 44 (mandibular first premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 45 (mandibular second premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first molar
Medium
👤 38M🦷 FDI 46
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 46 (mandibular first molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second molar
Hard
👤 43F🦷 FDI 47
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 47 (mandibular second molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary central incisor
Easy
👤 48M🦷 FDI 11
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 11 (maxillary central incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary lateral incisor
Easy
👤 53F🦷 FDI 12
53-year-old female presents with a draining gum boil but no pain. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary canine
Easy
👤 58M🦷 FDI 13
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 13 (maxillary canine). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first premolar
Medium
👤 63F🦷 FDI 14
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 14 (maxillary first premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second premolar
Medium
👤 68M🦷 FDI 15
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 15 (maxillary second premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 16 (maxillary first molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second molar
Hard
👤 18M🦷 FDI 17
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 17 (maxillary second molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular central incisor
Easy
👤 23F🦷 FDI 41
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 41 (mandibular central incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular lateral incisor
Easy
👤 28M🦷 FDI 42
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 42 (mandibular lateral incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular canine
Easy
👤 33F🦷 FDI 43
33-year-old female presents with a draining gum boil but no pain. Tooth 43 (mandibular canine). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first premolar
Medium
👤 38M🦷 FDI 44
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 44 (mandibular first premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second premolar
Medium
👤 43F🦷 FDI 45
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 45 (mandibular second premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first molar
Medium
👤 48M🦷 FDI 46
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 46 (mandibular first molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 47 (mandibular second molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary central incisor
Easy
👤 58M🦷 FDI 11
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 11 (maxillary central incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary lateral incisor
Easy
👤 63F🦷 FDI 12
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 12 (maxillary lateral incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary canine
Easy
👤 68M🦷 FDI 13
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 13 (maxillary canine). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first premolar
Medium
👤 73F🦷 FDI 14
73-year-old female presents with a draining gum boil but no pain. Tooth 14 (maxillary first premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second premolar
Medium
👤 18M🦷 FDI 15
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 15 (maxillary second premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first molar
Medium
👤 23F🦷 FDI 16
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 16 (maxillary first molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second molar
Hard
👤 28M🦷 FDI 17
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 17 (maxillary second molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 41 (mandibular central incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular lateral incisor
Easy
👤 38M🦷 FDI 42
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 42 (mandibular lateral incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular canine
Easy
👤 43F🦷 FDI 43
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 43 (mandibular canine). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first premolar
Medium
👤 48M🦷 FDI 44
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 44 (mandibular first premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second premolar
Medium
👤 53F🦷 FDI 45
53-year-old female presents with a draining gum boil but no pain. Tooth 45 (mandibular second premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first molar
Medium
👤 58M🦷 FDI 46
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 46 (mandibular first molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second molar
Hard
👤 63F🦷 FDI 47
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 47 (mandibular second molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary central incisor
Easy
👤 68M🦷 FDI 11
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 11 (maxillary central incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 12 (maxillary lateral incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary canine
Easy
👤 18M🦷 FDI 13
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 13 (maxillary canine). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first premolar
Medium
👤 23F🦷 FDI 14
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 14 (maxillary first premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second premolar
Medium
👤 28M🦷 FDI 15
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 15 (maxillary second premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first molar
Medium
👤 33F🦷 FDI 16
33-year-old female presents with a draining gum boil but no pain. Tooth 16 (maxillary first molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second molar
Hard
👤 38M🦷 FDI 17
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 17 (maxillary second molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular central incisor
Easy
👤 43F🦷 FDI 41
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 41 (mandibular central incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular lateral incisor
Easy
👤 48M🦷 FDI 42
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 42 (mandibular lateral incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 43 (mandibular canine). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first premolar
Medium
👤 58M🦷 FDI 44
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 44 (mandibular first premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second premolar
Medium
👤 63F🦷 FDI 45
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 45 (mandibular second premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first molar
Medium
👤 68M🦷 FDI 46
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 46 (mandibular first molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second molar
Hard
👤 73F🦷 FDI 47
73-year-old female presents with a draining gum boil but no pain. Tooth 47 (mandibular second molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary central incisor
Easy
👤 18M🦷 FDI 11
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 11 (maxillary central incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary lateral incisor
Easy
👤 23F🦷 FDI 12
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary canine
Easy
👤 28M🦷 FDI 13
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 13 (maxillary canine). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 14 (maxillary first premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second premolar
Medium
👤 38M🦷 FDI 15
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 15 (maxillary second premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first molar
Medium
👤 43F🦷 FDI 16
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 16 (maxillary first molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second molar
Hard
👤 48M🦷 FDI 17
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 17 (maxillary second molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular central incisor
Easy
👤 53F🦷 FDI 41
53-year-old female presents with a draining gum boil but no pain. Tooth 41 (mandibular central incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular lateral incisor
Easy
👤 58M🦷 FDI 42
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 42 (mandibular lateral incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular canine
Easy
👤 63F🦷 FDI 43
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 43 (mandibular canine). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first premolar
Medium
👤 68M🦷 FDI 44
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 44 (mandibular first premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 45 (mandibular second premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first molar
Medium
👤 18M🦷 FDI 46
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 46 (mandibular first molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second molar
Hard
👤 23F🦷 FDI 47
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 47 (mandibular second molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary central incisor
Easy
👤 28M🦷 FDI 11
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 11 (maxillary central incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary lateral incisor
Easy
👤 33F🦷 FDI 12
33-year-old female presents with a draining gum boil but no pain. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary canine
Easy
👤 38M🦷 FDI 13
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 13 (maxillary canine). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first premolar
Medium
👤 43F🦷 FDI 14
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 14 (maxillary first premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second premolar
Medium
👤 48M🦷 FDI 15
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 15 (maxillary second premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 16 (maxillary first molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second molar
Hard
👤 58M🦷 FDI 17
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 17 (maxillary second molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular central incisor
Easy
👤 63F🦷 FDI 41
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 41 (mandibular central incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular lateral incisor
Easy
👤 68M🦷 FDI 42
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 42 (mandibular lateral incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular canine
Easy
👤 73F🦷 FDI 43
73-year-old female presents with a draining gum boil but no pain. Tooth 43 (mandibular canine). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first premolar
Medium
👤 18M🦷 FDI 44
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 44 (mandibular first premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second premolar
Medium
👤 23F🦷 FDI 45
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 45 (mandibular second premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first molar
Medium
👤 28M🦷 FDI 46
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 46 (mandibular first molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 47 (mandibular second molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary central incisor
Easy
👤 38M🦷 FDI 11
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 11 (maxillary central incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary lateral incisor
Easy
👤 43F🦷 FDI 12
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 12 (maxillary lateral incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary canine
Easy
👤 48M🦷 FDI 13
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 13 (maxillary canine). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first premolar
Medium
👤 53F🦷 FDI 14
53-year-old female presents with a draining gum boil but no pain. Tooth 14 (maxillary first premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second premolar
Medium
👤 58M🦷 FDI 15
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 15 (maxillary second premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first molar
Medium
👤 63F🦷 FDI 16
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 16 (maxillary first molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second molar
Hard
👤 68M🦷 FDI 17
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 17 (maxillary second molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 41 (mandibular central incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular lateral incisor
Easy
👤 18M🦷 FDI 42
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 42 (mandibular lateral incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular canine
Easy
👤 23F🦷 FDI 43
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 43 (mandibular canine). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first premolar
Medium
👤 28M🦷 FDI 44
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 44 (mandibular first premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second premolar
Medium
👤 33F🦷 FDI 45
33-year-old female presents with a draining gum boil but no pain. Tooth 45 (mandibular second premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first molar
Medium
👤 38M🦷 FDI 46
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 46 (mandibular first molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second molar
Hard
👤 43F🦷 FDI 47
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 47 (mandibular second molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary central incisor
Easy
👤 48M🦷 FDI 11
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 11 (maxillary central incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 12 (maxillary lateral incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary canine
Easy
👤 58M🦷 FDI 13
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 13 (maxillary canine). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first premolar
Medium
👤 63F🦷 FDI 14
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 14 (maxillary first premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second premolar
Medium
👤 68M🦷 FDI 15
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 15 (maxillary second premolar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first molar
Medium
👤 73F🦷 FDI 16
73-year-old female presents with a draining gum boil but no pain. Tooth 16 (maxillary first molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second molar
Hard
👤 18M🦷 FDI 17
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 17 (maxillary second molar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular central incisor
Easy
👤 23F🦷 FDI 41
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 41 (mandibular central incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular lateral incisor
Easy
👤 28M🦷 FDI 42
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 42 (mandibular lateral incisor). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 43 (mandibular canine). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first premolar
Medium
👤 38M🦷 FDI 44
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 44 (mandibular first premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second premolar
Medium
👤 43F🦷 FDI 45
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 45 (mandibular second premolar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular first molar
Medium
👤 48M🦷 FDI 46
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 46 (mandibular first molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular second molar
Hard
👤 53F🦷 FDI 47
53-year-old female presents with a draining gum boil but no pain. Tooth 47 (mandibular second molar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary central incisor
Easy
👤 58M🦷 FDI 11
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 11 (maxillary central incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary lateral incisor
Easy
👤 63F🦷 FDI 12
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary canine
Easy
👤 68M🦷 FDI 13
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 13 (maxillary canine). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 14 (maxillary first premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second premolar
Medium
👤 18M🦷 FDI 15
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 15 (maxillary second premolar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary first molar
Medium
👤 23F🦷 FDI 16
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 16 (maxillary first molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary second molar
Hard
👤 28M🦷 FDI 17
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 17 (maxillary second molar). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular central incisor
Easy
👤 33F🦷 FDI 41
33-year-old female presents with a draining gum boil but no pain. Tooth 41 (mandibular central incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular lateral incisor
Easy
👤 38M🦷 FDI 42
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 42 (mandibular lateral incisor). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular canine
Easy
👤 43F🦷 FDI 43
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 43 (mandibular canine). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first premolar
Medium
👤 48M🦷 FDI 44
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 44 (mandibular first premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 45 (mandibular second premolar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular first molar
Medium
👤 58M🦷 FDI 46
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 46 (mandibular first molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular second molar
Hard
👤 63F🦷 FDI 47
63-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 47 (mandibular second molar). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary central incisor
Easy
👤 68M🦷 FDI 11
68-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 11 (maxillary central incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary lateral incisor
Easy
👤 73F🦷 FDI 12
73-year-old female presents with a draining gum boil but no pain. Tooth 12 (maxillary lateral incisor). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary canine
Easy
👤 18M🦷 FDI 13
18-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 13 (maxillary canine). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first premolar
Medium
👤 23F🦷 FDI 14
23-year-old female presents with no symptoms — found on routine radiographic review. Tooth 14 (maxillary first premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second premolar
Medium
👤 28M🦷 FDI 15
28-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 15 (maxillary second premolar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
33-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 16 (maxillary first molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary second molar
Hard
👤 38M🦷 FDI 17
38-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 17 (maxillary second molar). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful mandibular central incisor
Easy
👤 43F🦷 FDI 41
43-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 41 (mandibular central incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · mandibular lateral incisor
Easy
👤 48M🦷 FDI 42
48-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 42 (mandibular lateral incisor). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to mandibular canine
Easy
👤 53F🦷 FDI 43
53-year-old female presents with a draining gum boil but no pain. Tooth 43 (mandibular canine). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · mandibular first premolar
Medium
👤 58M🦷 FDI 44
58-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 44 (mandibular first premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · mandibular second premolar
Medium
👤 63F🦷 FDI 45
63-year-old female presents with no symptoms — found on routine radiographic review. Tooth 45 (mandibular second premolar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · mandibular first molar
Medium
👤 68M🦷 FDI 46
68-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 46 (mandibular first molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
73-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 47 (mandibular second molar). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive maxillary central incisor
Easy
👤 18M🦷 FDI 11
18-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 11 (maxillary central incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Painful maxillary lateral incisor
Easy
👤 23F🦷 FDI 12
23-year-old female presents with severe spontaneous throbbing pain, worse at night, and prolonged pain to cold lasting well beyond the stimulus. Tooth 12 (maxillary lateral incisor). Prolonged (lingering) response to cold. Mild tenderness to percussion. Extremely deep restoration close to the pulp on radiograph. No or minimal apical change.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Severe pulpitis + apical hypersensitivity. Spontaneous, lingering pain indicates Severe pulpitis (coronal pulp inflammation, possibly infected). Percussion sensitivity without a periapical radiolucency reflects apical hypersensitivity, not yet apical periodontitis. Management: pulpotomy (partial/full) or root canal treatment. (AAE/ESE 2025)
Management: Pulpotomy (partial or full) or root canal treatment, based on intraoperative assessment and restorability.
Biting pain · maxillary canine
Easy
👤 28M🦷 FDI 13
28-year-old male presents with no response to any stimulus and a dull ache on biting. Tooth 13 (maxillary canine). No response to thermal or electric pulp testing. Pain on percussion and palpation. Periapical radiolucency present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized symptomatic apical periodontitis. No response to pulp testing indicates Pulp necrosis. A periapical radiolucency of pulpal origin with pain to percussion/palpation = Localized symptomatic apical periodontitis. Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. Regenerative endodontic treatment if immature apex.
Swelling related to maxillary first premolar
Medium
👤 33F🦷 FDI 14
33-year-old female presents with a draining gum boil but no pain. Tooth 14 (maxillary first premolar). No response to pulp testing. No or mild pain to percussion. Periapical radiolucency extending to the cortical plate. A sinus tract traces to the tooth on a gutta-percha tracing radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized apical periodontitis with sinus tract. Necrotic pulp draining through a sinus tract that traces to the tooth is classified as Localized apical periodontitis with sinus tract in the 2025 system (replacing 'chronic apical abscess'). Endodontic intervention is indicated. (AAE/ESE 2025)
Management: Root canal treatment. The sinus tract typically resolves once the intracanal infection is eliminated.
Routine finding · maxillary second premolar
Medium
👤 38M🦷 FDI 15
38-year-old male presents with rapid facial swelling, fever and malaise over 24 hours. Tooth 15 (maxillary second premolar). No response to pulp testing. Diffuse facial swelling and facial asymmetry. Lymphadenopathy. Pain to percussion and palpation. Periapical radiolucency likely present.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + apical periodontitis with systemic involvement. Necrotic pulp with systemic signs (fever, diffuse facial swelling, lymphadenopathy) = Apical periodontitis with systemic involvement. Immediate intervention to drain infection, plus antibiotics and analgesics. (AAE/ESE 2025)
Management: Immediate endodontic intervention to drain infection. Antibiotics indicated. Pain medication as needed. Consider urgent referral if airway threatened.
Spontaneous pain · maxillary first molar
Medium
👤 43F🦷 FDI 16
43-year-old female presents with no symptoms — found on routine radiographic review. Tooth 16 (maxillary first molar). No response to pulp testing. No pain to percussion or palpation. A well-defined periapical radiolucency is present on the periapical radiograph.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Pulp necrosis + localized asymptomatic apical periodontitis. Necrotic pulp with a periapical radiolucency of pulpal origin but no symptoms = Localized asymptomatic apical periodontitis. Endodontic intervention is still indicated despite the absence of symptoms. (AAE/ESE 2025)
Management: Root canal treatment, even though asymptomatic — the lesion is of pulpal origin and will not resolve without intervention.
Gumline sensitivity · maxillary second molar
Hard
👤 48M🦷 FDI 17
48-year-old male presents with mild sensitivity at the gumline to cold and air, no spontaneous pain. Tooth 17 (maxillary second molar). Heightened, non-lingering response to cold localized to an area of gingival recession and exposed dentine. No percussion tenderness. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Hypersensitive pulp + clinically normal apical tissues. Exposed dentine (recession) causing a heightened but transient response, with an otherwise healthy pulp, defines Hypersensitive pulp in the 2025 system. No endodontic intervention — manage with desensitization/pulpal protection. (AAE/ESE 2025)
53-year-old female presents with pain only on biting, sharp and brief, with no thermal sensitivity. Tooth 41 (mandibular central incisor). Normal response to pulp testing. Moderate pain on percussion. Slight widening of the lamina dura, no frank apical radiolucency. History of recent high restoration.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Clinically normal pulp + apical hypersensitivity. A vital, normally responsive pulp with biting pain from hyper-occlusion (not pulpal inflammation) = Apical hypersensitivity. No endodontic intervention; adjust the occlusion. (AAE/ESE 2025)
Management: No endodontic intervention. Occlusal adjustment of the high restoration; review.
Cold-sensitive mandibular lateral incisor
Easy
👤 58M🦷 FDI 42
58-year-old male presents with sharp pain to cold and sweet that subsides within 1-2 seconds. Tooth 42 (mandibular lateral incisor). Heightened but non-lingering response to cold. No tenderness to percussion or palpation. Moderate caries; radiopaque dentine still visible between caries and pulp. No apical radiolucency.
What is the most accurate diagnosis? (AAE/ESE 2025)
Diagnosis: Mild pulpitis + clinically normal apical tissues. A non-lingering heightened cold response with caries approaching (but not reaching) the pulp, and no percussion tenderness, fits Mild pulpitis. Apical tissues are clinically normal. Management: restoration or vital pulp treatment depending on severity — root treatment unlikely needed. (AAE/ESE 2025)
Management: Restoration or selective caries removal; vital pulp treatment if exposure occurs. Root canal treatment unlikely required.
Diagnosis Tool
AAE/ESE 2025 Classification. Answer the prompts — the tool guides you to the current pulpal & periapical diagnosis with management.
1. Response to pulp testing (cold / EPT)?
2. Exposed dentine (recession / non-carious) rather than caries?
3. Spontaneous and/or severe pain (e.g. waking at night)?
4. Periapical & clinical findings?
5. Pain to percussion / biting (apical)?
Diagnosis · AAE/ESE 2025
PULPAL
—
PERIAPICAL
—
—
—
AAE/ESE 2025 — Quick Reference
🦷 Pulpal Diagnoses▼
Clinically normal pulp
Brief, normal response. No symptoms. No apical radiolucency.
→ No intervention.
Hypersensitive pulp
Heightened, non-lingering response from exposed dentine / recession. Healthy pulp.
→ Desensitize / pulpal protection.
Mild pulpitis
No–moderate symptoms; normal to heightened (non-lingering) response. Caries near but not into pulp.
→ Restoration or VPT. RCT unlikely.
Severe pulpitis
No–severe symptoms; may be spontaneous; lingering response. Bacterial invasion near pulp.
→ Pulpotomy (partial/full) or RCT.
Pulp necrosis
Loss of vitality; usually no response to testing. Apical radiolucency may be present.
→ RCT or regenerative endodontics.
Inconclusive pulp status
Unresponsive, uncertain (post-trauma, heavy restoration). No apical radiolucency.
→ Monitor for necrosis.
🔬 Periapical Diagnoses▼
Clinically normal apical tissues
No pain to percussion/palpation. Intact lamina dura, uniform PDL.
→ No intervention if pulp normal.
Apical hypersensitivity
Mild–moderate percussion pain, NOT of pulpal origin (e.g. hyper-occlusion). No/slight lamina dura widening.
→ No endo. Treat the cause.
Localized symptomatic apical periodontitis
Pulpal-origin inflammation. Pain to percussion/palpation. Radiolucency likely.
→ Endodontic intervention indicated.
Localized asymptomatic apical periodontitis
Pulpal-origin radiolucency. No/mild symptoms.
→ Endodontic intervention indicated.
Localized apical periodontitis with sinus tract
Pulpal-origin lesion to cortical plate. Sinus tract traces to tooth.
Summarized from the AAE/ESE 2025 Pulpal & Periapical Diagnosis Tables for clinical reference.
Dr. Endo — AI Assistant
🤖
Dr. Endo
Powered by Claude · EndoPedia Pro
Evidence-based only
Welcome. I'm Dr. Endo — your evidence-based endodontic clinical assistant.
Ask me anything: diagnosis, canal anatomy, instrumentation, irrigation, obturation, retreatment, or regenerative endo.
I answer only from Cohen's Pathways 11th & 12th Ed., AAE guidelines, JOE, IEJ, EEJ, ADA, and landmark peer-reviewed literature. I do not speculate beyond established evidence.
✓ Unlimited MCQs — full question bank
✓ Unlimited clinical cases
✓ Complete orifice atlas — all teeth
✓ All procedure guides
✓ Daily challenges & clinical tips
✓ Progress tracking & streaks
✓ Limited daily Dr. Endo AI
👑 Elite
$9.99/mo
✓ Everything in Scholar
✓ 🤖 Dr. Endo AI — unlimited* Evidence-graded answers from Cohen's, AAE, JOE, IEJ
✓ Priority access to new content *Generous fair-use daily allowance
Managed via App Store / Google Play · Cancel anytime
EndoPedia v2 · by Dr. Ahmed Khaled The Wikipedia of Endodontics
🔐
Admin Dashboard
Dr. Ahmed Khaled
Wrong password
Admin Panel
📋 Daily Facts
Add New Fact
⚡ Daily Challenge Questions
Add New Challenge
📝 MCQ Bank Questions
Add New MCQ
● Owner Console · Private
Dashboard
Dr. Ahmed Khaled · EndoPedia v2
Content Inventory
Quick Access
🗺️Atlas
📝MCQ Bank
🦷Cases
📋Guides
🔍Diagnose
⚙️Admin
Grow the Bank · toward 10,000
Imports save on this device and feed the no-repeat engine. To ship to all users, paste the exported JSON into the file (or send the batch to integrate).